Area of research
Physiology · Cellular and Molecular Neuroscience
Research interest
Research interests include Neuroscience, GABAA receptor, Pharmacology, Dopaminergic, Hyperalgesia, and Chronic pain.
Pharmacological activation of AMPK inhibits incision-evoked mechanical hypersensitivity and the development of hyperalgesic priming in mice
Piperidinyl thiazole isoxazolines: A new series of highly potent, slowly reversible FAAH inhibitors with analgesic properties
Neuroligin 2 regulates spinal GABAergic plasticity in hyperalgesic priming, a model of the transition from acute to chronic pain
Spinal Dopaminergic Projections Control the Transition to Pathological Pain Plasticity via a D<sub>1</sub>/D<sub>5</sub>-Mediated Mechanism
Inhibition of Carbonic Anhydrase Augments GABAA Receptor-Mediated Analgesia via a Spinal Mechanism of Action