Area of research
Surgery · Hepatology
Research interest
Research interests include Cholangiocarcinoma and Gallbladder Cancer Studies, Hepatocellular Carcinoma Treatment and Prognosis, Organ Transplantation Techniques and Outcomes, and Pancreatic and Hepatic Oncology Research.
Disrupted methionine cycle triggers muscle atrophy in cancer cachexia through epigenetic regulation of REDD1
Sparsentan ameliorates glomerular hypercellularity and inflammatory-gene networks induced by IgA1-IgG immune complexes in a mouse model of IgA nephropathy
γ-Linolenic acid in maternal milk drives cardiac metabolic maturation
Antioxidants stimulate BACH1-dependent tumor angiogenesis
LIF/JAK2/STAT1 Signaling Enhances Production of Galactose-Deficient IgA1 by IgA1-Producing Cell Lines Derived From Tonsils of Patients With IgA Nephropathy
Efficacy and safety of herbal medicines intervention for cachexia associated with cancer: A systematic review and meta‐analysis
Efficacy and Safety of Herbal Medicines Intervention for Cachexia Associated with Cancer: A Systematic Review and Meta-Analysis
The corepressors GPS2 and SMRT control enhancer and silencer remodeling via eRNA transcription during inflammatory activation of macrophages
Experimental evidence of pathogenic role of IgG autoantibodies in IgA nephropathy
Glomerular Immunodeposits of Patients with IgA Nephropathy Are Enriched for IgG Autoantibodies Specific for Galactose-Deficient IgA1
Hepatocyte-specific loss of GPS2 in mice reduces non-alcoholic steatohepatitis via activation of PPARα
Elucidation of the Mechanisms of Long-Distance mRNA Movement in a<i>Nicotiana benthamiana</i>/Tomato Heterograft System
GPS2 Deficiency Triggers Maladaptive White Adipose Tissue Expansion in Obesity via HIF1A Activation
Defining HIV-1 Envelope N-Glycan Microdomains through Site-Specific Heterogeneity Profiles
Loss of the co-repressor GPS2 sensitizes macrophage activation upon metabolic stress induced by obesity and type 2 diabetes
New Insights into the Pathogenesis of IgA Nephropathy
Cellular Signaling and Production of Galactose-Deficient IgA1 in IgA Nephropathy, an Autoimmune Disease