Area of research
Orthopedics and Sports Medicine · Molecular Biology
Research interest
Research interests include Internal medicine, Medicine, Endocrinology, Type 2 diabetes, Wnt signaling pathway, and Sclerostin.
Bone inflammation in postmenopausal women with type 2 diabetes or obesity in relation to Wnt signaling and bone strength
FMRP as a Key Regulator of Stress Granule Dynamics in Cholangiocarcinoma and Melanoma Cells
Fibre‐Enriched High‐Carbohydrate (FEHC) Diet Modulates Inflammation Without Affecting Bone Health in Older Women With Obesity: A Randomised Clinical Trial
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher advanced glycation end-products (AGEs) content and reduced bone strength
High-fiber diet reduces bone formation but does not affect bone microarchitecture in type 2 diabetes individuals
Increased bone inflammation in type 2 diabetes and obesity correlates with Wnt signaling downregulation and reduced bone strength
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher Advanced Glycation End-products (AGEs) content and reduced bone strength
Author Response: Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher Advanced Glycation End-products (AGEs) content and reduced bone strength
Author response: Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher advanced glycation end-products (AGEs) content and reduced bone strength
Increased bone inflammation in type 2 diabetes and obesity correlates with Wnt signaling downregulation and reduced bone strength
Increased bone inflammation in type 2 diabetes and obesity correlates with Wnt signaling downregulation and reduced bone strength
Oxidative Stress in Postmenopausal Women with or without Obesity
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher advanced glycation end-products (AGEs) content and reduced bone strength
Glycated haemoglobin in the first year after diagnosis of type 1 diabetes is an independent risk factor for diabetic retinopathy: The <scp>IMDIAB</scp> 25 years follow‐up study
Prevaccination Glucose Time in Range Correlates With Antibody Response to SARS-CoV-2 Vaccine in Type 1 Diabetes
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher Advanced Glycation End-products (AGEs) content and reduced bone strength
Wnt Signaling Regulation in Bone of Postmenopausal Women With Type 2 Diabetes
Changes in bone turnover markers in patients without bone metastases receiving immune checkpoint inhibitors: An exploratory analysis
Vertebral fractures and mortality risk in hospitalised patients during the COVID-19 pandemic emergency
Letter to the Editor From Viola et al: “Calcifediol Treatment and COVID-19-Related Outcomes”
Letter to the Editor from Viola et al.: “Diminishing Value from Multiple Serial Bone Densitometry in Women Receiving Antiresorptive Medication for Osteoporosis”
Sclerostin Regulation, Microarchitecture, and Advanced Glycation End-Products in the Bone of Elderly Women With Type 2 Diabetes
Gain-of-Function Lrp5 Mutation Improves Bone Mass and Strength and Delays Hyperglycemia in a Mouse Model of Insulin-Deficient Diabetes
Sclerostin Regulation, Microarchitecture, and Advanced Glycation End-Products in the Bone of Elderly Women With Type 2 Diabetes
Risk factors for fragility fractures in type 1 diabetes
Response to: Letter to editor in response to article published by Leanza et al.