Area of research
Pharmacology · Cellular and Molecular Neuroscience
Research interest
Research focused on Endocannabinoid system and Neuroscience, with related work in Inflammation, Lipid signaling, Immunology. Notable publications include 'Endocannabinoid signaling at the periphery: 50 years after THC', 'Bioactive Lipids and Chronic Inflammation: Managing the Fire Within', and 'Proresolving lipid mediators resolvin D1, resolvin D2, and maresin 1 are critical in modulating T cell responses'.
Bone inflammation in postmenopausal women with type 2 diabetes or obesity in relation to Wnt signaling and bone strength
Specialized pro-resolving mediators and autoimmunity: Recent insights and future perspectives
Fatty‐acid amide hydrolase inhibition mitigates Alzheimer's disease progression in mouse models of amyloidosis
Chronic palmitoylethanolamide administration via slow-release subcutaneous pellets promotes neuroprotection and mitigates neuroinflammation in the Tg2576 mouse model of Alzheimer’s disease
Probing Native CB<sub>2</sub> Receptor Mobility in Plasma Membranes of Living Cells by Fluorescence Recovery After Photobleaching
Fibre‐Enriched High‐Carbohydrate (FEHC) Diet Modulates Inflammation Without Affecting Bone Health in Older Women With Obesity: A Randomised Clinical Trial
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher advanced glycation end-products (AGEs) content and reduced bone strength
Cellular and Molecular Effects of Microgravity on the Immune System: A Focus on Bioactive Lipids
The endocannabinoid anandamide activates pro‐resolving pathways in human primary macrophages by engaging both <scp> CB <sub>2</sub> </scp> and <scp>GPR18</scp> receptors
Simulated Microgravity Affects Pro-Resolving Properties of Primary Human Monocytes
Interaction between α-Synuclein and Bioactive Lipids: Neurodegeneration, Disease Biomarkers and Emerging Therapies
Protective effects of fatty acid amide hydrolase inhibition in UVB-activated microglia
Increased bone inflammation in type 2 diabetes and obesity correlates with Wnt signaling downregulation and reduced bone strength
Beyond Symptom Management: FAAH Inhibition as a Path to Mitigate Alzheimer’s Disease Progression in Mouse Models of Amyloidosis
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher Advanced Glycation End-products (AGEs) content and reduced bone strength
Author Response: Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher Advanced Glycation End-products (AGEs) content and reduced bone strength
Author response: Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher advanced glycation end-products (AGEs) content and reduced bone strength
Increased bone inflammation in type 2 diabetes and obesity correlates with Wnt signaling downregulation and reduced bone strength
Increased bone inflammation in type 2 diabetes and obesity correlates with Wnt signaling downregulation and reduced bone strength
Bioactive signalling lipids as drivers of chronic liver diseases
Oxidative Stress in Postmenopausal Women with or without Obesity
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher advanced glycation end-products (AGEs) content and reduced bone strength
The specialized pro-resolving lipid mediator Protectin D1 affects macrophages differentiation and activity in Adult-onset Still’s disease and COVID-19, two hyperinflammatory diseases sharing similar transcriptomic profiles
Dysregulation of Resolvin E1 Metabolism and Signaling in a Light-Damage Model of Age-Related Macular Degeneration
Computational and Experimental Drug Repurposing of FDA-Approved Compounds Targeting the Cannabinoid Receptor CB1
A game of networks: Do different lipids interact to orchestrate inflammatory homeostasis?
Bone canonical Wnt signaling is downregulated in type 2 diabetes and associates with higher Advanced Glycation End-products (AGEs) content and reduced bone strength
POS0237 ADULT-ONSET STILL’S DISEASE AND COVID 19 SHARE GENETIC EXPRESSION RELATED TO HYPERINFLAMMATION: DEFINING THE POSSIBLE ROLE OF THE SPECIALIZED PRO-RESOLVING LIPID MEDIATOR PROTECTIN D1 IN MODULATING MACROPHAGES POLARIZATION AND FUNCTION
Wnt Signaling Regulation in Bone of Postmenopausal Women With Type 2 Diabetes
Detection of cannabinoid receptor type 2 in native cells and zebrafish with a highly potent, cell-permeable fluorescent probe