Area of research
Molecular Biology · Nephrology
Research interest
Research interests include Ion Transport and Channel Regulation, Renal function and acid-base balance, Electrolyte and hormonal disorders, and Ion channel regulation and function.
The proximal tubule through an NBCe1-dependent mechanism regulates collecting duct phenotypic and remodeling responses to acidosis.
Acid-base effects of combined renal deletion of NBCe1-A and NBCe1-B.
Expression of lactate dehydrogenase A and B isoforms in the mouse kidney
Expression of lactate dehydrogenase A and B isoforms in the mouse kidney.
Role of the renal androgen receptor in sex differences in ammonia metabolism.
The Renal Physiology of Pendrin-Positive Intercalated Cells
The Renal Physiology of Pendrin-Positive Intercalated Cells.
Differences in renal BMAL1 contribution to Na<sup>+</sup> homeostasis and blood pressure control in male and female mice.
Aldosterone Regulates Pendrin and Epithelial Sodium Channel Activity through Intercalated Cell Mineralocorticoid Receptor–Dependent and –Independent Mechanisms over a Wide Range in Serum Potassium
Testosterone modulates renal ammonia metabolism.
NBCe1-A is required for the renal ammonia and K<sup>+</sup> response to hypokalemia.
Differences in acidosis-stimulated renal ammonia metabolism in the male and female kidney.
Podocyte-specific expression of Cre recombinase promotes glomerular basement membrane thickening
Podocyte-specific expression of Cre recombinase promotes glomerular basement membrane thickening.
Regulation of renal NaDC1 expression and citrate excretion by NBCe1-A.
The Role of Intercalated Cell Nedd4–2 in BP Regulation, Ion Transport, and Transporter Expression
Transcriptomes of major renal collecting duct cell types in mouse identified by single-cell RNA-seq
Pendrin gene ablation alters ENaC subcellular distribution and open probability
Effect of dietary protein restriction on renal ammonia metabolism
Pendrin localizes to the adrenal medulla and modulates catecholamine release
Proximal tubule H-ferritin mediates iron trafficking in acute kidney injury