Area of research
Infectious Diseases · Epidemiology
Research interest
Research interests include Tuberculosis Research and Epidemiology, Mycobacterium research and diagnosis, HIV Research and Treatment, and Infectious Diseases and Tuberculosis.
Antigen-specific B cells direct T follicular-like helper cells into lymphoid follicles to mediate Mycobacterium tuberculosis control
Data from Long-term <i>In vitro</i> Expansion Alters the Biology of Adult Mesenchymal Stem Cells
Data from Long-term <i>In vitro</i> Expansion Alters the Biology of Adult Mesenchymal Stem Cells
Supplementary Figure 2A from Long-term <i>In vitro</i> Expansion Alters the Biology of Adult Mesenchymal Stem Cells
The immunoregulatory landscape of human tuberculosis granulomas
Diagnosis of paediatric tuberculosis by optically detecting two virulence factors on extracellular vesicles in blood samples
Human M1 macrophages express unique innate immune response genes after mycobacterial infection to defend against tuberculosis
Developing tuberculosis vaccines for people with HIV: consensus statements from an international expert panel
BNT162b vaccines protect rhesus macaques from SARS-CoV-2
A non-canonical type 2 immune response coordinates tuberculous granuloma formation and epithelialization
Visualizing the dynamics of tuberculosis pathology using molecular imaging
Medical imaging of pulmonary disease in SARS-CoV-2-exposed non-human primates
Lung Epithelial Signaling Mediates Early Vaccine-Induced CD4 <sup>+</sup> T Cell Activation and <i>Mycobacterium tuberculosis</i> Control
Pulmonary Mycobacterium tuberculosis control associates with CXCR3- and CCR6-expressing antigen-specific Th1 and Th17 cell recruitment
Chronic Immune Activation in TB/HIV Co-infection
Antiretroviral therapy does not reduce tuberculosis reactivation in a tuberculosis-HIV coinfection model
Formation of Lung Inducible Bronchus Associated Lymphoid Tissue Is Regulated by Mycobacterium tuberculosis Expressed Determinants
Group 3 innate lymphoid cells mediate early protective immunity against tuberculosis
Mechanisms of reactivation of latent tuberculosis infection due to SIV coinfection
IMPACT-TB*: A Phase II Trial Assessing the Capacity of Low Dose Imatinib to Induce Myelopoiesis and Enhance Host Anti-Microbial Immunity Against Tuberculosis. *Imatinib Mesylate per Oral As a Clinical Therapeutic for TB
In vivo inhibition of tryptophan catabolism reorganizes the tuberculoma and augments immune-mediated control of<i>Mycobacterium tuberculosis</i>
CD4 <sup>+</sup> T-cell–independent mechanisms suppress reactivation of latent tuberculosis in a macaque model of HIV coinfection
S100A8/A9 Proteins Mediate Neutrophilic Inflammation and Lung Pathology during Tuberculosis