Area of research
Infectious Diseases · Immunology
Research interest
Research interests include Tuberculosis Research and Epidemiology, Mycobacterium research and diagnosis, Immunodeficiency and Autoimmune Disorders, and Psoriasis: Treatment and Pathogenesis.
β-Glucan reprograms neutrophils to promote disease tolerance against influenza A virus
BCG vaccination alters the epigenetic landscape of progenitor cells in human bone marrow to influence innate immune responses
Vaccination with Mincle agonist UM-1098 and mycobacterial antigens induces protective Th1 and Th17 responses
Antigen-specific B cells direct T follicular-like helper cells into lymphoid follicles to mediate Mycobacterium tuberculosis control
Rifampicin drug resistance and host immunity in tuberculosis: more than meets the eye
The immunoregulatory landscape of human tuberculosis granulomas
Mitochondrial cyclophilin D promotes disease tolerance by licensing NK cell development and IL-22 production against influenza virus
Lung Epithelial Signaling Mediates Early Vaccine-Induced CD4 <sup>+</sup> T Cell Activation and <i>Mycobacterium tuberculosis</i> Control
Trained immunity, tolerance, priming and differentiation: distinct immunological processes
COVID-19: viral–host interactome analyzed by network based-approach model to study pathogenesis of SARS-CoV-2 infection
Chronic Immune Activation in TB/HIV Co-infection
Antiretroviral therapy does not reduce tuberculosis reactivation in a tuberculosis-HIV coinfection model
Formation of Lung Inducible Bronchus Associated Lymphoid Tissue Is Regulated by Mycobacterium tuberculosis Expressed Determinants
Group 3 innate lymphoid cells mediate early protective immunity against tuberculosis
Targeting innate immunity for tuberculosis vaccination
Mechanisms of reactivation of latent tuberculosis infection due to SIV coinfection
In vivo inhibition of tryptophan catabolism reorganizes the tuberculoma and augments immune-mediated control of<i>Mycobacterium tuberculosis</i>
Itaconate Links Inhibition of Succinate Dehydrogenase with Macrophage Metabolic Remodeling and Regulation of Inflammation
CD4 <sup>+</sup> T-cell–independent mechanisms suppress reactivation of latent tuberculosis in a macaque model of HIV coinfection
Targeting dendritic cells to accelerate T-cell activation overcomes a bottleneck in tuberculosis vaccine efficacy
<i>Mycobacterium tuberculosis</i> Impairs Dendritic Cell Functions through the Serine Hydrolase Hip1
S100A8/A9 Proteins Mediate Neutrophilic Inflammation and Lung Pathology during Tuberculosis
Interleukin-17-dependent CXCL13 mediates mucosal vaccine–induced immunity against tuberculosis
Variants in toll-like receptor 9 gene influence susceptibility to tuberculosis in a Mexican population