J. Craig Venter Institute · US
Area of research
Hematology · Molecular Biology
Research interest
Research interests include Acute Myeloid Leukemia Research, Protein Degradation and Inhibitors, Epigenetics and DNA Methylation, and Nanoplatforms for cancer theranostics.
Blood cell parameters and risk of nonalcoholic fatty liver disease: a comprehensive Mendelian randomization study
Exploring the role of alternative lengthening of telomere-related genes in diagnostic modeling for non-alcoholic fatty liver disease
N6-methyladenosine demethylase FTO impairs hepatic ischemia–reperfusion injury via inhibiting Drp1-mediated mitochondrial fragmentation
Bio-inspired imager improves sensitivity in near-infrared fluorescence image-guided surgery
UCP2 Mitigates the Loss of Human Spermatozoa Motility by Promoting mROS Elimination
Optical See-Through Cancer Vision Goggles Enable Direct Patient Visualization and Real-Time Fluorescence-Guided Oncologic Surgery
Mitochondrial Uncoupling Protein 2 in human cumulus cells is associated with regulating autophagy and apoptosis, maintaining gap junction integrity and progesterone synthesis
Oridonin induces apoptosis through the mitochondrial pathway in human gastric cancer SGC-7901 cells
DOT1L inhibits SIRT1-mediated epigenetic silencing to maintain leukemic gene expression in MLL-rearranged leukemia
Binocular Goggle Augmented Imaging and Navigation System provides real-time fluorescence image guidance for tumor resection and sentinel lymph node mapping
Inactivation of Eed impedes MLL-AF9–mediated leukemogenesis through Cdkn2a-dependent and Cdkn2a-independent mechanisms in a murine model
Real-Time Fluorescence Image-Guided Oncologic Surgery
Single camera imaging system for color and near-infrared fluorescence image guided surgery
Information-optimal Scalable Compressive Imaging System
Notch pathway activation targets AML-initiating cell homeostasis and differentiation
Chromatin-modifying enzymes as modulators of reprogramming
Polycomb repressive complex 2 is required for MLL-AF9 leukemia
STAT5 Is Crucial to Maintain Leukemic Stem Cells in Acute Myelogenous Leukemias Induced by MOZ-TIF2