Carsten Willam

S3-Leitlinie Nierenersatztherapie in der Intensivmedizin

Curriculum „Internistische Intensivmedizin und Notfallmedizin“

Definition of the Post-COVID syndrome using a symptom-based Post-COVID score in a prospective, multi-center, cross-sectoral cohort of the German National Pandemic Cohort Network (NAPKON)

Curriculum Klinische Akut- und Notfallmedizin – Schwerpunkt Innere Medizin

Reconfiguration and loss of peritubular capillaries in chronic kidney disease

Inhibition of transforming growth factor β1 signaling in resident interstitial cells attenuates profibrotic gene expression and preserves erythropoietin production during experimental kidney fibrosis in mice

Mononuclear phagocytes orchestrate prolyl hydroxylase inhibition-mediated renoprotection in chronic tubulointerstitial nephritis

HIF-1α promotes cyst progression in a mouse model of autosomal dominant polycystic kidney disease

Activation of Hypoxia Signaling in Stromal Progenitors Impairs Kidney Development

Myeloid Cell–Derived HIF-1α Promotes Control of <i>Leishmania major</i>

Erythropoietin production by PDGFR-β+ cells

Ferritin-Mediated Iron Sequestration Stabilizes Hypoxia-Inducible Factor-1α upon LPS Activation in the Presence of Ample Oxygen

Identification of Plasma Metabolites Prognostic of Acute Kidney Injury after Cardiac Surgery with Cardiopulmonary Bypass

Hypoxia-Inducible Factor-1α Causes Renal Cyst Expansion through Calcium-Activated Chloride Secretion

Deletion of von Hippel–Lindau Protein Converts Renin-Producing Cells into Erythropoietin-Producing Cells

Cystic kidney diseases

Analysis of human urine reveals metabolic changes related to the development of acute kidney injury following cardiac surgery