Area of research
Surgery · Cardiology and Cardiovascular Medicine
Research interest
Research interests include Biomedical and Chemical Research, Renin-Angiotensin System Studies, Medical Practices and Rehabilitation, and Connexins and lens biology.
Enhancing the safety of ciltacabtagene autoleucel in relapsed multiple myeloma (MM): Identification of potentially modifiable risk-factors associated with delayed neurotoxicity and non-relapse mortality
Investigation of the Role of Connexins During Skin Wound Healing in a Novel 3D-In-Vivo-Wound Healing Model
Impact of extramedullary multiple myeloma on outcomes with idecabtagene vicleucel
Idecabtagene vicleucel chimeric antigen receptor T-cell therapy for relapsed/refractory multiple myeloma with renal impairment
Intact prostaglandin signaling through EP2 and EP4 receptors in stromal progenitor cells is required for normal development of the renal cortex in mice
Localization and characterization of proenkephalin-A as a potential biomarker for kidney disease in murine and human kidneys
Inhibition of transforming growth factor β1 signaling in resident interstitial cells attenuates profibrotic gene expression and preserves erythropoietin production during experimental kidney fibrosis in mice
Localization of angiotensin II type 1 receptor gene expression in rodent and human kidneys
Prolyl‐4‐hydroxylases 2 and 3 control erythropoietin production in renin‐expressing cells of mouse kidneys
Connexin mRNA distribution in adult mouse kidneys
Endothelin receptors in renal interstitial cells do not contribute to the development of fibrosis during experimental kidney disease
Different subpopulations of kidney interstitial cells produce erythropoietin and factors supporting tissue oxygenation in response to hypoxia in vivo
Connexin37‐Dependent Mechanisms Selectively Contribute to Modulate Angiotensin II‐Mediated Hypertension
Apparently normal kidney development in mice with conditional disruption of ANG II-AT<sub>1</sub>receptor genes in FoxD1-positive stroma cell precursors
Angiotensin II Short-Loop Feedback
Lack of connexin 40 decreases the calcium sensitivity of renin-secreting juxtaglomerular cells
Activation of Hypoxia Signaling in Stromal Progenitors Impairs Kidney Development
Inducible deletion of connexin 40 in adult mice causes hypertension and disrupts pressure control of renin secretion
Connexin 43 is not essential for the control of renin synthesis and secretion
Development of renal renin-expressing cells does not involve PDGF-B-PDGFR-<i>β</i>signaling
Control of renin secretion from kidneys with renin cell hyperplasia
Distribution and functional relevance of connexins in renin-producing cells