Area of research
Molecular Biology · Cellular and Molecular Neuroscience
Research interest
Research interests include Cell biology, Vascular smooth muscle, Angiogenesis, Biology, Endothelial stem cell, and Medicine.
Endothelial cells drive organ fibrosis in mice by inducing expression of the transcription factor SOX9
Endothelial Heterogeneity in the Response to Autophagy Drives Small Vessel Muscularization in Pulmonary Hypertension
Nuclear factor of activated T-cells 5 is indispensable for a balanced adaptive transcriptional response of lung endothelial cells to hypoxia
Endothelial Notch1 signaling in white adipose tissue promotes cancer cachexia
NFAT5/TonEBP Limits Pulmonary Vascular Resistance in the Hypoxic Lung by Controlling Mitochondrial Reactive Oxygen Species Generation in Arterial Smooth Muscle Cells
Loss of Nfat5 promotes lipid accumulation in vascular smooth muscle cells
Loss of the serine protease HTRA1 impairs smooth muscle cells maturation
Expansion of functional personalized cells with specific transgene combinations
Genetic ablation of NFAT5/TonEBP in smooth muscle cells impairs flow‐ and pressure‐induced arterial remodeling in mice
Abstract 450: Vascular Smooth Muscle Cell-expressed Tie2 Controls Atherosclerosis Progression
Loss of Mpdz impairs ependymal cell integrity leading to perinatal‐onset hydrocephalus in mice
Endothelial cell spheroids as a versatile tool to study angiogenesis <i>in vitro</i>
Transdermal drug targeting and functional imaging of tumor blood vessels in the mouse auricle
Abstract 441: Rgs5 Controls Myogenic Responses of Vascular Smooth Muscle Cells
The tetraspanin network modulates MT1-MMP cell surface trafficking
RGS5 is crucial for vascular smooth muscle cells during arterial remodeling in mice
No Evidence for a Functional Role of Bi-Directional Notch Signaling during Angiogenesis
Angiopoietin-1 mediates inhibition of hypertension-induced release of angiopoietin-2 from endothelial cells
Impact of the EphrinB/EphB system on pro‐inflammatory monocyte‐endothelial cell interaction