Alberto Bosque

EZH2 inhibition mitigates HIV immune evasion, reduces reservoir formation, and promotes skewing of CD8+ T cells toward less-exhausted phenotypes

The latency-reversing agent HODHBt synergizes with IL-15 to enhance cytotoxic function of HIV-specific T cells

SARS CoV-2 mRNA vaccination exposes latent HIV to Nef-specific CD8+ T-cells

BCL-2 antagonism sensitizes cytotoxic T cell–resistant HIV reservoirs to elimination ex vivo

Targeting Cellular and Tissue HIV Reservoirs With Toll-Like Receptor Agonists

Long non-coding RNAs and latent HIV – A search for novel targets for latency reversal

Latent HIV reservoirs exhibit inherent resistance to elimination by CD8+ T cells

Dual TLR2 and TLR7 agonists as HIV latency-reversing agents

SAMHD1 Phosphorylation Coordinates the Anti-HIV-1 Response by Diverse Interferons and Tyrosine Kinase Inhibition

Benzotriazoles Reactivate Latent HIV-1 through Inactivation of STAT5 SUMOylation

T-cell responses targeting HIV Nef uniquely correlate with infected cell frequencies after long-term antiretroviral therapy

Microbiota promotes systemic T-cell survival through suppression of an apoptotic factor

HIV Integration Site Analysis of Cellular Models of HIV Latency with a Probe-Enriched Next-Generation Sequencing Assay

Comparative proteomics of exosomes secreted by tumoral Jurkat T cells and normal human T cell blasts unravels a potential tumorigenic role for valosin-containing protein

BIRC2/cIAP1 Is a Negative Regulator of HIV-1 Transcription and Can Be Targeted by Smac Mimetics to Promote Reversal of Viral Latency

<i>Ex Vivo</i>Bioactivity and HIV-1 Latency Reversal by Ingenol Dibenzoate and Panobinostat in Resting CD4⁺T Cells from Aviremic Patients

Modeling HIV-1 Latency in Primary T Cells Using a Replication-Competent Virus

An HIV-Encoded Antisense Long Noncoding RNA Epigenetically Regulates Viral Transcription

Ruxolitinib and Tofacitinib Are Potent and Selective Inhibitors of HIV-1 Replication and Virus Reactivation <i>In Vitro</i>

Downmodulation of CCR7 by HIV-1 Vpu Results in Impaired Migration and Chemotactic Signaling within CD4+ T Cells

An In-Depth Comparison of Latent HIV-1 Reactivation in Multiple Cell Model Systems and Resting CD4+ T Cells from Aviremic Patients

Reactivation of latent HIV-1 in central memory CD4+T cells through TLR-1/2 stimulation

HIV latency and integration site placement in five cell-based models

Kinase Control of Latent HIV-1 Infection: PIM-1 Kinase as a Major Contributor to HIV-1 Reactivation

Cyclin T1 and CDK9 T-Loop Phosphorylation Are Downregulated during Establishment of HIV-1 Latency in Primary Resting Memory CD4⁺T Cells

Kinase Control Prevents HIV-1 Reactivation in Spite of High Levels of Induced NF-κB Activity