Frank Wolschendorf

A copper-dependent compound restores ampicillin sensitivity in multidrug-resistant Staphylococcus aureus

High-throughput screening and Bayesian machine learning for copper-dependent inhibitors of <i>Staphylococcus aureus</i>

Assaying for Inorganic Polyphosphate in Bacteria

Differential Susceptibility of Mycoplasma and Ureaplasma Species to Compound-Enhanced Copper Toxicity

Copper and Antibiotics

Mycobacterium tuberculosis exploits the PPM1A signaling pathway to block host macrophage apoptosis

8-Hydroxyquinolines Are Boosting Agents of Copper-Related Toxicity in Mycobacterium tuberculosis

Protein Phosphatase, Mg2+/Mn2+-dependent 1A controls the innate antiviral and antibacterial response of macrophages during HIV-1 and <i>Mycobacterium tuberculosis</i> infection

A Macrophage Infection Model to Predict Drug Efficacy Against <i>Mycobacterium Tuberculosis</i>

Combinatorial phenotypic screen uncovers unrecognized family of extended thiourea inhibitors with copper-dependent anti-staphylococcal activity

Targeting Biofilm Associated &lt;em&gt;Staphylococcus aureus&lt;/em&gt; Using Resazurin Based Drug-susceptibility Assay

Mycobacteria, metals, and the macrophage

Disulfiram and Copper Ions Kill Mycobacterium tuberculosis in a Synergistic Manner

Stable Phenotypic Changes of the Host T Cells Are Essential to the Long-Term Stability of Latent HIV-1 Infection

Copper Complexation Screen Reveals Compounds with Potent Antibiotic Properties against Methicillin-Resistant Staphylococcus aureus

Porins Increase Copper Susceptibility of Mycobacterium tuberculosis

Kinase Control of Latent HIV-1 Infection: PIM-1 Kinase as a Major Contributor to HIV-1 Reactivation

An AP-1 Binding Site in the Enhancer/Core Element of the HIV-1 Promoter Controls the Ability of HIV-1 To Establish Latent Infection

Copper-Boosting Compounds: a Novel Concept for Antimycobacterial Drug Discovery

Kinase Control Prevents HIV-1 Reactivation in Spite of High Levels of Induced NF-κB Activity

Selected Drugs with Reported Secondary Cell-Differentiating Capacity Prime Latent HIV-1 Infection for Reactivation