Area of research
Molecular Biology · Physiology
Research interest
Research interests include Chemistry, Endocrinology, Internal medicine, Distal convoluted tubule, Stimulation, and Angiotensin II.
Dapagliflozin inhibits ferroptosis and ameliorates renal fibrosis in diabetic C57BL/6J mice
Kir4.1/Kir5.1 of distal convoluted tubule is required for short-term angiotensin-II-induced stimulation of Na-Cl cotransporter
[The mechanism of blood pressure regulation by high potassium diet in the kidney].
PubMed 2022cited by 2position: middle
PGF<sub>2α</sub> stimulates the 10-pS Cl<sup>−</sup> channel and thiazide-sensitive Na<sup>+</sup>-Cl<sup>−</sup> cotransporter in the distal convoluted tubule
Kir4.1/Kir5.1 Activity in the DCT is Essential for Angiotensin II Induced Stimulation of Thiazide‐Sensitive NCC Expression/Activity.
Inhibition of AT2R and Bradykinin Type II Receptor (BK2R) Compromises High K <sup>+</sup> Intake-Induced Renal K <sup>+</sup> Excretion
Bradykinin Stimulates Renal Na <sup>+</sup> and K <sup>+</sup> Excretion by Inhibiting the K <sup>+</sup> Channel (Kir4.1) in the Distal Convoluted Tubule
Norepinephrine-Induced Stimulation of Kir4.1/Kir5.1 Is Required for the Activation of NaCl Transporter in Distal Convoluted Tubule
AT2R (Angiotensin II Type 2 Receptor)-Mediated Regulation of NCC (Na-Cl Cotransporter) and Renal K Excretion Depends on the K Channel, Kir4.1
[The function and regulation of basolateral Kir4.1 and Kir4.1/Kir5.1 in renal tubules].
PubMed 2018cited by 0position: last
Disruption of KCNJ10 (Kir4.1) stimulates the expression of ENaC in the collecting duct
Effects of adenosine stimulation on the mRNA expression of CLCNKB in the basolateral medullary thick ascending limb of the rat kidney
Vasopressin-induced stimulation of the Na+-activated K+ channels is responsible for maintaining the basolateral K+ conductance of the thick ascending limb (TAL) in EAST/SeSAME syndrome
ADH-induced Stimulation of Na-activated K Channels is Responsible for Maintaining Basolateral K Conductance of the Thick Ascending Limb (TAL) in EAST/Sesame Syndrome
Angiotensin II Stimulates Basolateral 10-pS Cl Channels in the Thick Ascending Limb
Angiotensin II stimulates basolateral 50-pS K channels in the thick ascending limb
Angiotensin II stimulates basolateral 10 pS Cl channels, a main type of Cl channels in the thick ascending limb (TAL)
Insulin-like growth factor-1 (IGF-1) inhibits the basolateral Cl channels in the thick ascending limb of the rat kidney