Xinxin Meng

Kir4.1/Kir5.1 of distal convoluted tubule is required for short-term angiotensin-II-induced stimulation of Na-Cl cotransporter

YOLOv7-based attention and improved feature fusion small object detection algorithm

Deep hierarchical multiscale attention networks for image super-resolution

MFDF-YOLOv7: YOLOv7-Based Multiscale Feature Dynamic Fusion Small Object Detection Algorithm

PGF_2α stimulates the 10-pS Cl⁻ channel and thiazide-sensitive Na⁺-Cl⁻ cotransporter in the distal convoluted tubule

Kir4.1/Kir5.1 Activity in the DCT is Essential for Angiotensin II Induced Stimulation of Thiazide‐Sensitive NCC Expression/Activity.

Inhibition of AT2R and Bradykinin Type II Receptor (BK2R) Compromises High K ⁺ Intake-Induced Renal K ⁺ Excretion

Bradykinin Stimulates Renal Na ⁺ and K ⁺ Excretion by Inhibiting the K ⁺ Channel (Kir4.1) in the Distal Convoluted Tubule

Norepinephrine-Induced Stimulation of Kir4.1/Kir5.1 Is Required for the Activation of NaCl Transporter in Distal Convoluted Tubule

[The function and regulation of basolateral Kir4.1 and Kir4.1/Kir5.1 in renal tubules].

Vasopressin-induced stimulation of the Na+-activated K+ channels is responsible for maintaining the basolateral K+ conductance of the thick ascending limb (TAL) in EAST/SeSAME syndrome

ADH-induced Stimulation of Na-activated K Channels is Responsible for Maintaining Basolateral K Conductance of the Thick Ascending Limb (TAL) in EAST/Sesame Syndrome