Rainer Schreiber

Anoctamin 9 determines Ca2+ signals during activation of T-lymphocytes

Dystonia caused by ANO3 variants is due to attenuated Ca2+ influx by ORAI1

Case Report of Pediatric HPCA-Associated Dystonia: Analysis of Ca2+ and K+ Channel Dynamics and Experience With Pallidal Deep Brain Stimulation

Inhibition of mucus secretion by niclosamide and benzbromarone in airways and intestine

Epithelial Anoctamins

Anoctamins in epithelial transport

A novel SLC26A9 inhibitor, S9-A13, reveals a minor contribution of SLC26A9 to constitutive airway chloride secretion

Functional Interdependence of Anoctamins May Influence Conclusions from Overexpression Studies

Niclosamide, but not ivermectin, inhibits anoctamin 1 and 6 and attenuates inflammation of the respiratory tract

The Anion Channel TMEM16a/Ano1 Modulates CFTR Activity, but Does Not Function as an Apical Anion Channel in Colonic Epithelium from Cystic Fibrosis Patients and Healthy Individuals

Broadening the clinical spectrum: molecular mechanisms and new phenotypes of <i>ANO3</i>-dystonia

SLC26A9 in airways and intestine: secretion or absorption?

TMEM16A/F support exocytosis but do not inhibit Notch-mediated goblet cell metaplasia of BCi-NS1.1 human airway epithelium

KCNE1 does not shift TMEM16A from a Ca2+ dependent to a voltage dependent Cl- channel and is not expressed in renal proximal tubule

Pathogenic Relationships in Cystic Fibrosis and Renal Diseases: CFTR, SLC26A9 and Anoctamins

Pathogenic Relationships in Cystic Fibrosis: CFTR, SLC26A9 and Anoctamins

Airway Delivery of Hydrogel-Encapsulated Niclosamide for the Treatment of Inflammatory Airway Disease

The <scp>SLC26A9</scp> inhibitor <scp>S9‐A13</scp> provides no evidence for a role of <scp>SLC26A9</scp> in airway chloride secretion but suggests a contribution to regulation of <scp>ASL pH</scp> and gastric proton secretion

Pharmacological inhibitors of the cystic fibrosis transmembrane conductance regulator exert off-target effects on epithelial cation channels

Influence of Anoctamin-4 and -9 on ADAM10 and ADAM17 Sheddase Function

Expression of SLC26A9 in Airways and Its Potential Role in Asthma

A <scp>TMEM16J</scp> variant leads to dysregulated cytosolic calcium which may lead to renal disease

Systems Approaches to Unravel Molecular Function: High-content siRNA Screen Identifies TMEM16A Traffic Regulators as Potential Drug Targets for Cystic Fibrosis

Paneth Cell Secretion in vivo Requires Expression of Tmem16a and Tmem16f

P. aeruginosa Induced Lipid Peroxidation Causes Ferroptotic Cell Death in Airways

CLCA1 Regulates Airway Mucus Production and Ion Secretion Through TMEM16A

The molecular mechanism of CFTR‐ and secretin‐dependent renal bicarbonate excretion

Mucus Release and Airway Constriction by TMEM16A May Worsen Pathology in Inflammatory Lung Disease

Gender-Dependent Phenotype in Polycystic Kidney Disease Is Determined by Differential Intracellular Ca2+ Signals

IRAG2 Interacts with IP3-Receptor Types 1, 2, and 3 and Regulates Intracellular Ca2+ in Murine Pancreatic Acinar Cells