Area of research
Immunology
Research interest
Research interests include Immune Cell Function and Interaction, IL-33, ST2, and ILC Pathways, T-cell and B-cell Immunology, and Psoriasis: Treatment and Pathogenesis.
Nutrition impact on ILC3 maintenance and function centers on a cell-intrinsic CD71-iron axis.
Kindlin-2 inhibits TNF/NF-κB-Caspase 8 pathway in hepatocytes to maintain liver development and function
Bcl11b sustains multipotency and restricts effector programs of intestinal-resident memory CD8<sup>+</sup> T cells.
Group 3 innate lymphoid cell pyroptosis represents a host defence mechanism against Salmonella infection.
Kynurenine induces T cell fat catabolism and has limited suppressive effects in vivo
Mitochondrial transcription factor A in RORγt<sup>+</sup> lymphocytes regulate small intestine homeostasis and metabolism.
Ahr-Foxp3-RORγt axis controls gut homing of CD4<sup>+</sup> T cells by regulating GPR15.
Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation
IL-17–producing ST2+ group 2 innate lymphoid cells play a pathogenic role in lung inflammation
The Aryl Hydrocarbon Receptor Preferentially Marks and Promotes Gut Regulatory T Cells
BCL-2 protects human and mouse Th17 cells from glucocorticoid-induced apoptosis
Ikaros Inhibits Group 3 Innate Lymphoid Cell Development and Function by Suppressing the Aryl Hydrocarbon Receptor Pathway
Commensal bacteria protect against food allergen sensitization
Induction of Innate Lymphoid Cell-Derived Interleukin-22 by the Transcription Factor STAT3 Mediates Protection against Intestinal Infection
Group 3 Innate Lymphoid Cells Inhibit T-Cell-Mediated Intestinal Inflammation through Aryl Hydrocarbon Receptor Signaling and Regulation of Microflora
RETRACTED ARTICLE: Suppression of inflammation and acute lung injury by Miz1 via repression of C/EBP-δ