Area of research
Nephrology · Pediatrics, Perinatology and Child Health
Research interest
Research interests include Chronic Kidney Disease and Diabetes, Renal Diseases and Glomerulopathies, Neonatal and fetal brain pathology, and Thyroid Disorders and Treatments.
Circular RNA METTL9 contributes to neuroinflammation following traumatic brain injury by complexing with astrocytic SND1
Empagliflozin alleviates atherosclerotic calcification by inhibiting osteogenic differentiation of vascular smooth muscle cells
Mesenchymal Stem Cell-Derived Exosomes: Toward Cell-Free Therapeutic Strategies in Chronic Kidney Disease
Multiplex Assessment of Serum Chemokines CCL2, CCL5, CXCL1, CXCL10, and CXCL13 Following Traumatic Brain Injury
Faecal Microbiota Transplantation and Chronic Kidney Disease
[Effect of Rehmanniae Radix Praeparata on energy metabolism in prefrontal cortex of rats with attention deficit hyperactivity disorder based on "static Yin and dynamic Yang" theory].
Metformin Attenuates Renal Fibrosis in a Mouse Model of Adenine-Induced Renal Injury Through Inhibiting TGF-β1 Signaling Pathways
Hyperbaric oxygen therapy improves neurological function via the p38-MAPK/CCL2 signaling pathway following traumatic brain injury
RIPK3 blockade attenuates tubulointerstitial fibrosis in a mouse model of diabetic nephropathy
RIPK3: A New Player in Renal Fibrosis
Bergaptol from blossoms of <i>Citrus aurantium</i> L. var. <i>amara</i> Engl inhibits LPS-induced inflammatory responses and ox-LDL-induced lipid deposition
Inhibitory effects of multi-components from Gynostemma pentaphyllum (Thunb.) Makino on macrophage foam cell formation exhibit multi-target characteristics
Gypenoside LVI attenuates foam cell formation by promoting cholesterol export and inhibiting inflammation response
Polyphenols from Blossoms of <i>Citrus aurantium</i> L. var. <i>amara</i> Engl. Show Significant Anti-Complement and Anti-Inflammatory Effects
Thioredoxin interacting protein (TXNIP) regulates tubular autophagy and mitophagy in diabetic nephropathy through the mTOR signaling pathway
KCa3.1 mediates dysfunction of tubular autophagy in diabetic kidneys via PI3k/Akt/mTOR signaling pathways
Thioredoxin-interacting protein mediates dysfunction of tubular autophagy in diabetic kidneys through inhibiting autophagic flux
Inhibition of Kidney Proximal Tubular Glucose Reabsorption Does Not Prevent against Diabetic Nephropathy in Type 1 Diabetic eNOS Knockout Mice
Blockade of KCa3.1 Ameliorates Renal Fibrosis Through the TGF-β1/Smad Pathway in Diabetic Mice
KCa3.1 mediates activation of fibroblasts in diabetic renal interstitial fibrosis