Area of research
Immunology · Public Health, Environmental and Occupational Health
Research interest
Research interests include Acute Lymphoblastic Leukemia research, Immunotherapy and Immune Responses, Immune cells in cancer, and Immunodeficiency and Autoimmune Disorders.
Epigenetic remodeling via HDAC6 inhibition amplifies anti-tumoral immune responses in myeloid leukemia cells.
A gut instinct for childhood leukemia prevention: microbiome-targeting recommendations aimed at parents and caregivers
H1‐0 is a specific mediator of the repressive ETV6::RUNX1 transcriptional landscape in preleukemia and B cell acute lymphoblastic leukemia
H1-0 is a specific mediator of the repressive ETV6::RUNX1 transcriptional landscape in preleukemia and B cell acute lymphoblastic leukemia.
Deep transfer learning approach for automated cell death classification reveals novel ferroptosis-inducing agents in subsets of B-ALL
Immune training enhances anti-viral responses and improves outcomes in Pax5<sup>-/+</sup> mice susceptible to chronic infection.
Deep transfer learning approach for automated cell death classification reveals novel ferroptosis-inducing agents in subsets of B-ALL.
Modulation of the <i>ETV6::RUNX1</i> Gene Fusion Prevalence in Newborns by Corticosteroid Use During Pregnancy.
Epigenetic remodeling via HDAC6 inhibition amplifies anti-tumoral immune responses in myeloid leukemia cells
Serum-free differentiation platform for the generation of B lymphocytes and natural killer cells from human CD34+ cord blood progenitors
Supplementary Figure 2 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Table 3 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Figure7 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Figure 6 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Figure 3 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary materials and methods from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Figure 5 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Figure 1 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Table 1 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Figure 4 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
Supplementary Table 2 from BAFF Attenuates Immunosuppressive Monocytes in the Melanoma Tumor Microenvironment
A brain organoid/ALL coculture model reveals the AP-1 pathway as critically associated with CNS involvement of BCP-ALL.
Optical genome mapping identifies structural variants in potentially new cancer predisposition candidate genes in pediatric cancer patients.
A gut instinct for childhood leukemia prevention: microbiome-targeting recommendations aimed at parents and caregivers.
Maternal Lifestyle and Prenatal Risk Factors for Childhood Leukemia: A Review of the Existing Evidence.
Hyperdiploid acute lymphoblastic leukemia in children with <i>LZTR1</i> germline variants.
Proteogenomic profiling uncovers differential therapeutic vulnerabilities between TCF3::PBX1 and TCF3::HLF translocated B-cell acute lymphoblastic leukemia
Proteogenomic profiling uncovers differential therapeutic vulnerabilities between <i>TCF3::PBX1</i> and <i>TCF3::HLF</i> translocated B-cell acute lymphoblastic leukemia.
Linker histone H1-0 is a specific mediator of the repressive ETV6::RUNX1 transcriptional landscape
Monogenic early-onset lymphoproliferation and autoimmunity: Natural history of STAT3 gain-of-function syndrome.