Area of research
Public Health, Environmental and Occupational Health · Hematology
Research interest
Research interests include Acute Lymphoblastic Leukemia research, Acute Myeloid Leukemia Research, Cancer-related molecular mechanisms research, and Epigenetics and DNA Methylation.
Proteomic profiling reveals ACSS2 facilitating metabolic support in acute myeloid leukemia
FAT1 expression in T-cell acute lymphoblastic leukemia (T-ALL) modulates proliferation and WNT signaling
Actionable loss of SLF2 drives B‐cell lymphomagenesis and impairs the DNA damage response
Activated SUMOylation restricts MHC class I antigen presentation to confer immune evasion in cancer
Small-molecule SUMO inhibition for biomarker-informed B-cell lymphoma therapy
An alternative CYB5A transcript is expressed in aneuploid ALL and enriched in relapse
CXCR4 hyperactivation cooperates with TCL1 in CLL development and aggressiveness
PAX5 biallelic genomic alterations define a novel subgroup of B-cell precursor acute lymphoblastic leukemia
Long non-coding RNAs defining major subtypes of B cell precursor acute lymphoblastic leukemia
Integrated analysis of relapsed B-cell precursor Acute Lymphoblastic Leukemia identifies subtype-specific cytokine and metabolic signatures
Long non-coding RNAs defining major subtypes of B cell precursor acute lymphoblastic leukemia
Acute myeloid leukemia in the elderly is characterized by a distinct genetic and epigenetic landscape
Multi-Omics of Adult T Cell Acute Lymphoblastic Leukemia
Homocygous PAX5Sequence Mutations Define a Novel Subtype of B Cell Precursor Acute Lymphoblastic Leukemia
Molecular alterations in bone marrow mesenchymal stromal cells derived from acute myeloid leukemia patients
Silencing of GATA3 defines a novel stem cell-like subgroup of ETP-ALL
Multi-Genomics of Relapsed B-Cell Precursor Acute Lymphoblastic Leukemia Reveals Three Distinct Genetic Clusters Characterized By Different Alterations
Acute Myeloid Leukemia in the Elderly Is Characterized By a Distinct Genetic Landscape
Molecular Alterations in Refractory Acute Myeloid Leukemia and Persistent Preleukemic Lesions Are Disclosed Using Mesenchymal Stromal Cells As Germline Control
Molecular Alterations in Bone Marrow Mesenchymal Stroma Cells of AML Patients