Area of research
Hepatology · Epidemiology
Research interest
Research interests include Oxidative stress, Liver regeneration, Biology, Internal medicine, Lipotoxicity, and Endocrinology.
Bile acid-induced apoptosis and bile acid synthesis are reduced by over-expression of Augmenter of Liver Regeneration (ALR) in a STAT3-dependent mechanism
Bile acids down-regulate the expression of Augmenter of Liver Regeneration (ALR) via SHP/HNF4α1 and independent of Egr-1
Attenuated lipotoxicity and apoptosis is linked to exogenous and endogenous augmenter of liver regeneration by different pathways
Reduced HNF3b (FOXA2) levels in steatosis leads to diminished expression of anti-lipoapoptotic protein ALR
Hepatic detoxification and anti-oxidative response in liver tissue with cholestasis: analysis of Nrf2 dependent gene expression
Nrf2 Activates Augmenter of Liver Regeneration (ALR) via Antioxidant Response Element and Links Oxidative Stress to Liver Regeneration
Nrf2 regulates antioxidative and liver regeneration promoting genes