Area of research
Epidemiology · Hepatology
Research interest
Research interests include Biology, Internal medicine, Hepatocyte, Cell biology, Liver regeneration, and Medicine.
Cell type specific decoding of TGF-β2 production, signaling and outcome in the dynamics of cholestatic liver disease
Hepatic Bone Morphogenetic Protein and Activin Membrane-Bound Inhibitor Levels Decline in Hepatitis C but Are Not Associated with Progression of Hepatocellular Carcinoma
Gas6 in chronic liver disease—a novel blood-based biomarker for liver fibrosis
Delineating signals of TGF-β2 expression induction and consequences of its signalling in cholestatic liver disease-mediated cholangiopathies
Preclinical Efficacy and Toxicity Analysis of the Pan-Histone Deacetylase Inhibitor Gossypol for the Therapy of Colorectal Cancer or Hepatocellular Carcinoma
Chemerin Overexpression in the Liver Protects against Inflammation in Experimental Non-Alcoholic Steatohepatitis
Hepatocyte expressed chemerin-156 does not protect from experimental non-alcoholic steatohepatitis
Correction to: Toxicogenomics directory of chemically exposed human hepatocytes
TGF-β2 silencing to target biliary-derived liver diseases
Inflammation-associated suppression of metabolic gene networks in acute and chronic liver disease
Antifibrotic Effects of Amyloid-Beta and Its Loss in Cirrhotic Liver
Entwicklung und Funktion der Leber
Entwicklung und Funktion der Leber
Augmenter of liver regeneration: Essential for growth and beyond
Bile acid-induced apoptosis and bile acid synthesis are reduced by over-expression of Augmenter of Liver Regeneration (ALR) in a STAT3-dependent mechanism
Bile acids down-regulate the expression of Augmenter of Liver Regeneration (ALR) via SHP/HNF4α1 and independent of Egr-1
Soluble Axl is an accurate biomarker of cirrhosis and hepatocellular carcinoma development: results from a large scale multicenter analysis
Attenuated lipotoxicity and apoptosis is linked to exogenous and endogenous augmenter of liver regeneration by different pathways
Transforming Growth Factor β1 (TGF-β1) Activates Hepcidin mRNA Expression in Hepatocytes
Gene networks and transcription factor motifs defining the differentiation of stem cells into hepatocyte-like cells
FK866-induced NAMPT inhibition activates AMPK and downregulates mTOR signaling in hepatocarcinoma cells
Smad7 regulates compensatory hepatocyte proliferation in damaged mouse liver and positively relates to better clinical outcome in human hepatocellular carcinoma
Corrigendum to “Gene networks and transcription factor motifs defining the differentiation of human stem cells into hepatocyte-like cells” [J Hepatol 2015;63:934–942]
Gene networks and transcription factor motifs defining the differentiation of stem cells into hepatocyte-like cells
Toxicogenomics directory of chemically exposed human hepatocytes
Regulation and function of the atypical cadherin FAT1 in hepatocellular carcinoma
Adiponectin Isoforms Differentially Affect Gene Expression and the Lipidome of Primary Human Hepatocytes
Attenuated and Protease-Profile Modified Sendai Virus Vectors as a New Tool for Virotherapy of Solid Tumors
Recent advances in 2D and 3D in vitro systems using primary hepatocytes, alternative hepatocyte sources and non-parenchymal liver cells and their use in investigating mechanisms of hepatotoxicity, cell signaling and ADME
Resveratrol as a Pan-HDAC Inhibitor Alters the Acetylation Status of Jistone Proteins in Human-Derived Hepatoblastoma Cells