Area of research
Molecular Biology · Genetics
Research interest
Research interests include Biology, Colitis, Immunology, Inflammatory bowel disease, Medicine, and Dysbiosis.
Transplant of microbiota from Crohn’s disease patients to germ-free mice results in colitis
Infection and inflammation stimulate expansion of a CD74+ Paneth cell subset to regulate disease progression
Dynamics of dark fermentation microbial communities in the light of lactate and butyrate production
Non-canonical NRF2 activation promotes a pro-diabetic shift in hepatic glucose metabolism
Intestinal Regulatory T Cells
Dynamics of Gut Microbiota Recovery after Antibiotic Exposure in Young and Old Mice (A Pilot Study)
Paneth Cell-Derived Lysozyme Defines the Composition of Mucolytic Microbiota and the Inflammatory Tone of the Intestine
Intestinal Epithelial Expression of MHCII Determines Severity of Chemical, T-Cell–Induced, and Infectious Colitis in Mice
Emerging Roles of Disabled Homolog 2 (DAB2) in Immune Regulation
Elevating EGFR-MAPK program by a nonconventional Cdc42 enhances intestinal epithelial survival and regeneration
Rapid Downregulation of DAB2 by Toll-Like Receptor Activation Contributes to a Pro-Inflammatory Switch in Activated Dendritic Cells
Total CD3 T Cells Are Necessary and Sufficient to Induce Colitis in Immunodeficient Mice With Dendritic Cell–Specific Deletion of TGFbR2: A Novel IBD Model to Study CD4 and CD8 T-Cell Interaction
Microbial dysbiosis associated with impaired intestinal Na+/H+ exchange accelerates and exacerbates colitis in ex-germ free mice
Transforming Growth Factor Beta Signaling in Dendritic Cells Is Required for Immunotolerance to Sperm in the Epididymis
SLC9 Gene Family: Function, Expression, and Regulation
Vitamins and Minerals in Inflammatory Bowel Disease
Physiology of Intestinal Absorption and Secretion
Pathophysiology of Intestinal Na+/H+ Exchange
Transcriptional Reprogramming and Resistance to Colonic Mucosal Injury in Poly(ADP-ribose) Polymerase 1 (PARP1)-deficient Mice
Reduced Epithelial Na+/H+ Exchange Drives Gut Microbial Dysbiosis and Promotes Inflammatory Response in T Cell-Mediated Murine Colitis
Expression of Ca<sub>v</sub>1.3 calcium channel in the human and mouse colon: posttranscriptional inhibition by IFNγ
The Role of Curcumin in Modulating Colonic Microbiota During Colitis and Colon Cancer Prevention
Gut Microbial Dysbiosis May Predict Diarrhea and Fatigue in Patients Undergoing Pelvic Cancer Radiotherapy: A Pilot Study
T Lymphocyte Dynamics in Inflammatory Bowel Diseases: Role of the Microbiome
Epithelial Transport in Inflammatory Bowel Diseases
Clinical Characteristics Associated With Postoperative Intestinal Epithelial Barrier Dysfunction in Children With Congenital Heart Disease*
Reduced colonic microbial diversity is associated with colitis in NHE3-deficient mice
Post-Translational Loss of Renal TRPV5 Calcium Channel Expression, Ca2+ Wasting, and Bone Loss in Experimental Colitis
Dendritic Cell-Specific Disruption of TGF-β Receptor II Leads to Altered Regulatory T Cell Phenotype and Spontaneous Multiorgan Autoimmunity
Polyclonal CD4+Foxp3+ Treg cells induce TGFβ-dependent tolerogenic dendritic cells that suppress the murine lupus-like syndrome