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Peter Winter

Broad Institute · US
Area of research
Plant Science · Molecular Biology
Research interest
Research interests include Genetic and Environmental Crop Studies, Legume Nitrogen Fixing Symbiosis, Single-cell and spatial transcriptomics, and Agricultural pest management studies.
h-index
52
citations
11,043
works
238
NIH funding
primary concept
Biology
email

Recent publications

Improving atlas-scale single-cell annotation models with hierarchical cross-entropy loss.
2026cited by 0position: contributordoi
Beyond alignment: synergistic integration is required for multimodal cell foundation models
2026cited by 0position: contributordoi
Adapting systems biology to address the complexity of human disease in the single-cell era
Nature Reviews Genetics 2025cited by 10position: middledoi
Adapting systems biology to address the complexity of human disease in the single-cell era.
2025cited by 5position: contributordoi
Hierarchical cross-entropy loss improves atlas-scale single-cell annotation models
2025cited by 1position: contributordoi
Supplementary Data from Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and Extensive Intratumoral Heterogeneity
2025cited by 0position: contributordoi
Supplementary Data from Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and Extensive Intratumoral Heterogeneity
2025cited by 0position: contributordoi
Data from Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and Extensive Intratumoral Heterogeneity
2025cited by 0position: contributordoi
Adaptive resampling for improved machine learning in imbalanced single-cell datasets
2025cited by 0position: contributordoi
Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
Cancer Discovery 2024cited by 224position: middledoi
Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer.
2024cited by 180position: contributordoi
Cancer tissue of origin constrains the growth and metabolism of metastases
Nature Metabolism 2024cited by 32position: middledoi
Scalable, compressed phenotypic screening using pooled perturbations
Nature Biotechnology 2024cited by 16position: middledoi
Mutation and cell state compatibility is required and targetable in Ph <i>+</i> acute lymphoblastic leukemia minimal residual disease
bioRxiv (Cold Spring Harbor Laboratory) 2024cited by 4position: firstdoi
Mutation and cell state compatibility is required and targetable in Ph <i>+</i> acute lymphoblastic leukemia minimal residual disease
2024cited by 4position: contributordoi
A knockoff calibration method to avoid over-clustering in single-cell RNA-sequencing
2024cited by 1position: contributordoi
Evaluating the role of pre-training dataset size and diversity on single-cell foundation model performance
2024cited by 1position: contributordoi
Supplemental Table 5 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Figures 1-11 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 1 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Scalable nonparametric clustering with unified marker gene selection for single-cell RNA-seq data
2024cited by 0position: contributordoi
Data from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 2 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 7 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 8 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 3 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 4 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 6 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Supplemental Table 9 from Mechanisms of Resistance to Oncogenic KRAS Inhibition in Pancreatic Cancer
2024cited by 0position: contributordoi
Spatially Resolved Single-Cell Assessment of Pancreatic Cancer Expression Subtypes Reveals Co-expressor Phenotypes and Extensive Intratumoral Heterogeneity.
2023cited by 84position: contributordoi

Grants

No grants ingested yet.

Frequent collaborators

· 10 papers (2023–2026)Ava P. Amini · Microsoft Research (United Kingdom)8 papers (2024–2026)Lorin Crawford · Yale University8 papers (2024–2026)Srivatsan Raghavan · Dana-Farber Brigham Cancer Center6 papers (2024–2026) · 4 papers (2013–2015)Alex K. Shalek · Broad Institute4 papers (2019–2025)Alex K. Shalek · Massachusetts Institute of Technology4 papers (2024–2025) · 4 papers (2024–2024)Sören Müller · Gene Therapy Laboratory4 papers (2013–2015)Günter Kahl · Goethe University Frankfurt3 papers (2014–2015) · 3 papers (2024–2024)Ina Koch · Goethe University Frankfurt3 papers (2014–2015)Fabian Afonso-Grunz · GenXPro (Germany)3 papers (2014–2015)Tyler Jacks · Howard Hughes Medical Institute2 papers (2019–2022)Philipp Bruns · Technical University of Munich2 papers (2015–2015)Fabian J. Theis · University of Lübeck2 papers (2015–2015)Christoph Michalski · Heidelberg University2 papers (2015–2015)Bo Kong · Heidelberg University2 papers (2015–2015) · 2 papers (2015–2015)Sebastiano Cultrera di Montesano · Broad Institute2 papers (2025–2026)