Area of research
Surgery · Molecular Biology
Research interest
Research interests include Internal medicine, Endocrinology, Insulin, Glucose homeostasis, Biology, and Insulin resistance.
Sex Differences in Pancreatic β-Cell Physiology and Glucose Homeostasis in C57BL/6J Mice
Deconstructing the integrated oscillator model for pancreatic <mml:math xmlns:mml="http://www.w3.org/1998/Math/MathML" altimg="si237.svg" display="inline" id="d1e1207"><mml:mi>β</mml:mi></mml:math>-cells
K<sub>ATP</sub> channel activity and slow oscillations in pancreatic beta cells are regulated by mitochondrial ATP production
Ca<sup>2+</sup> release or Ca<sup>2+</sup> entry, that is the question: what governs Ca<sup>2+</sup> oscillations in pancreatic β cells?
Oscillations in K(ATP) conductance drive slow calcium oscillations in pancreatic β-cells
Pulsatile Basal Insulin Secretion Is Driven by Glycolytic Oscillations
Slow oscillations persist in pancreatic beta cells lacking phosphofructokinase M
<i>Chop</i> / <i>Ddit3</i> depletion in β cells alleviates ER stress and corrects hepatic steatosis in mice
Symbiosis of Electrical and Metabolic Oscillations in Pancreatic β-Cells
Multiple Feedback Mechanisms Underlying Beta Cell Secretory Oscillations
IAPP toxicity activates HIF1α/PFKFB3 signaling delaying β-cell loss at the expense of β-cell function
Gestational exposure to metformin programs improved glucose tolerance and insulin secretion in adult male mouse offspring
Closing in on the Mechanisms of Pulsatile Insulin Secretion
Overexpression of Kinase-Dead mTOR Impairs Glucose Homeostasis by Regulating Insulin Secretion and Not β-Cell Mass
Upregulation of an inward rectifying K+ channel can rescue slow Ca2+ oscillations in K(ATP) channel deficient pancreatic islets
Phase Analysis of Metabolic Oscillations and Membrane Potential in Pancreatic Islet β -Cells
Ca 2+ Effects on ATP Production and Consumption Have Regulatory Roles on Oscillatory Islet Activity
Pulsatile insulin secretion, impaired glucose tolerance and type 2 diabetes
Leptin-inhibited PBN neurons enhance responses to hypoglycemia in negative energy balance
Calcium and Metabolic Oscillations in Pancreatic Islets: Who's Driving the Bus?
Electrical, Calcium, and Metabolic Oscillations in Pancreatic Islets
Slow oscillations of K<sub>ATP</sub> conductance in mouse pancreatic islets provide support for electrical bursting driven by metabolic oscillations
Electrical, Calcium, and Metabolic Oscillations in Pancreatic Islets
Phosphofructo-2-kinase/Fructose-2,6-bisphosphatase Modulates Oscillations of Pancreatic Islet Metabolism