Milan Raška

Loss of GalNAc-T14 links O-glycosylation defects to alterations in B cell homing in IgA nephropathy

Galactose-Deficient IgA1 B cells in the Circulation of IgA Nephropathy Patients Carry Preferentially Lambda Light Chains and Mucosal Homing Receptors

Leukemia Inhibitory Factor Signaling Enhances Production of Galactose-Deficient IgA1 in IgA Nephropathy

Role of Epstein-Barr Virus in Pathogenesis and Racial Distribution of IgA Nephropathy

Secretory IgA N-glycans contribute to the protection against E. coli O55 infection of germ-free piglets

Glycan Positioning Impacts HIV-1 Env Glycan-Shield Density, Function, and Recognition by Antibodies

Defining HIV-1 Envelope N-Glycan Microdomains through Site-Specific Heterogeneity Profiles

Glucocorticoids Reduce Aberrant O-Glycosylation of IgA1 in IgA Nephropathy Patients

Inhibition of STAT3 Signaling Reduces IgA1 Autoantigen Production in IgA Nephropathy

Lasioglossins LLIII affect the morphogenesis of <i>Candida albicans</i> and reduces the duration of experimental vaginal candidiasis in mice

The Origin and Activities of IgA1-Containing Immune Complexes in IgA Nephropathy

Multi-layered nanofibrous mucoadhesive films for buccal and sublingual administration of drug-delivery and vaccination nanoparticles - important step towards effective mucosal vaccines

Liposomal nanocarriers for plasminogen activators

IgA nephropathy enigma

Somatic Mutations Modulate Autoantibodies against Galactose-Deficient IgA1 in IgA Nephropathy

The Position of His-Tag in Recombinant OspC and Application of Various Adjuvants Affects the Intensity and Quality of Specific Antibody Response after Immunization of Experimental Mice

Endotoxin-minimized HIV-1 p24 fused to murine hsp70 activates dendritic cells, facilitates endocytosis and p24-specific Th1 response in mice

IgA Nephropathy and Related Diseases

Cytokines Alter IgA1 O-Glycosylation by Dysregulating C1GalT1 and ST6GalNAc-II Enzymes

Differential glycosylation of envelope gp120 is associated with differential recognition of HIV-1 by virus-specific antibodies and cell infection

Enzymatic Sialylation of IgA1 O-Glycans: Implications for Studies of IgA Nephropathy

N-Acetylgalactosaminide α2,6-sialyltransferase II is a candidate enzyme for sialylation of galactose-deficient IgA1, the key autoantigen in IgA nephropathy

HIV-1 Envelope Glycan Moieties Modulate HIV-1 Transmission

Humoral Immune Responses to <scp>HIV</scp> in the Mucosal Secretions and Sera of <scp>HIV</scp>‐Infected Women

IgA Nephropathy: Molecular Mechanisms of the Disease

Aberrant O-glycosylation and anti-glycan antibodies in an autoimmune disease IgA nephropathy and breast adenocarcinoma