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Julie H. Cox

Southwestern Medical Center · US
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Area of research
Biomedical Engineering · Physiology
Research interest
Research topics from publications: Overexpression of Long-Chain Acyl-CoA Synthetase 5 Increases Fatty Acid Oxidation and Free Radical Formation While Attenuating Insulin Signaling in Primary Human Skeletal Myotubes. Representative work: In rodent skeletal muscle, acyl-coenzyme A (CoA) synthetase 5 (ACSL-5) is suggested to localize to the mitochondria but its precise function in human skeletal muscle is unknown. The purpose of these studies was to define the role of ACSL-5 in mitochondrial fatty acid metabolism and the potential effects on insulin action in human skeletal muscle cells (HSKMC). Primary myoblasts isolated from vastus lateralis (obese women (body mass index (BMI) = 34.7 ± 3.1 kg/m2)) were transfected with ACSL-5 plasmid DNA or green fluorescent protein (GFP) vector (control), differentiated into myotubes, and harvested (7 days). HSKMC were assayed for complete and incomplete fatty acid oxidation ([1-14C] palmit
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Recent publications

Overexpression of Long-Chain Acyl-CoA Synthetase 5 Increases Fatty Acid Oxidation and Free Radical Formation While Attenuating Insulin Signaling in Primary Human Skeletal Myotubes
International Journal of Environmental Research and Public Health 2019cited by 21position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

· 1 papers (2019–2019) · 1 papers (2019–2019)Robert C. Hickner · University of Iowa1 papers (2019–2019)Hyo‐Bum Kwak · University of Tennessee at Knoxville1 papers (2019–2019) · 1 papers (2019–2019)P. Darrell Neufer · Wake Forest University1 papers (2019–2019)
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