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Stefan F. Lichtenthaler

Technical University of Munich · DE
Area of research
Physiology · Neurology
Research interest
Research interests include Biology, Cell biology, Neuroscience, ADAM10, Receptor, and Pathogenesis.
h-index
citations
2,192
works
26
NIH funding
primary concept
email

Recent publications

Multicenter Longitudinal Quality Assessment of MS-Based Proteomics in Plasma and Serum
Journal of Proteome Research 2025cited by 11position: middledoi
Multicenter evaluation of label-free quantification in human plasma on a high dynamic range benchmark set
Nature Communications 2025cited by 6position: middledoi
A unified classification approach rating clinical utility of protein biomarkers across neurologic diseases
EBioMedicine 2023cited by 30position: middledoi
Deciphering sources of PET signals in the tumor microenvironment of glioblastoma at cellular resolution
Science Advances 2023cited by 21position: middledoi
Deciphering sources of PET signals in the tumor microenvironment of glioblastoma at cellular resolution
bioRxiv (Cold Spring Harbor Laboratory) 2023cited by 2position: middledoi
ADAM10 and ADAM17 promote SARS‐CoV‐2 cell entry and spike protein‐mediated lung cell fusion
EMBO Reports 2022cited by 126position: lastdoi
Experimental evidence for temporal uncoupling of brain Aβ deposition and neurodegenerative sequelae
Nature Communications 2022cited by 27position: middledoi
Signatures of glial activity can be detected in the CSF proteome
Proceedings of the National Academy of Sciences 2022cited by 23position: middledoi
Proteolytically generated soluble Tweak Receptor Fn14 is a blood biomarker for γ‐secretase activity
EMBO Molecular Medicine 2022cited by 12position: lastdoi
The case for low-level BACE1 inhibition for the prevention of Alzheimer disease
Nature Reviews Neurology 2021cited by 133position: lastdoi
Highly efficient intercellular spreading of protein misfolding mediated by viral ligand-receptor interactions
Nature Communications 2021cited by 69position: middledoi
Shed CNTNAP2 ectodomain is detectable in CSF and regulates Ca2+ homeostasis and network synchrony via PMCA2/ATP2B2
Neuron 2021cited by 43position: middledoi
Fibroblast Growth Factor 2‐Mediated Regulation of Neuronal Exosome Release Depends on VAMP3/Cellubrevin in Hippocampal Neurons
Advanced Science 2020cited by 70position: middledoi
Cell-type-specific profiling of brain mitochondria reveals functional and molecular diversity
Nature Neuroscience 2019cited by 332position: middledoi
Degradome of soluble ADAM10 and ADAM17 metalloproteases
Cellular and Molecular Life Sciences 2019cited by 65position: middledoi
NrCAM is a marker for substrate‐selective activation of ADAM10 in Alzheimer's disease
EMBO Molecular Medicine 2019cited by 54position: lastdoi
BACE2 distribution in major brain cell types and identification of novel substrates
Life Science Alliance 2018cited by 72position: middledoi
BACE1 Physiological Functions May Limit Its Use as Therapeutic Target for Alzheimer's Disease
Trends in Neurosciences 2016cited by 169position: middledoi
Seizure protein 6 and its homolog seizure 6-like protein are physiological substrates of BACE1 in neurons
Molecular Neurodegeneration 2016cited by 122position: lastdoi
Generation of aggregation prone N-terminally truncated amyloid β peptides by meprin β depends on the sequence specificity at the cleavage site
Molecular Neurodegeneration 2016cited by 72position: middledoi
Function, therapeutic potential and cell biology of <scp>BACE</scp> proteases: current status and future prospects
Journal of Neurochemistry 2014cited by 311position: lastdoi
Dietary (−)-epicatechin as a potent inhibitor of βγ-secretase amyloid precursor protein processing
Neurobiology of Aging 2014cited by 87position: middledoi
The FTLD risk factor TMEM106B and MAP6 control dendritic trafficking of lysosomes
The EMBO Journal 2013cited by 170position: middledoi
Chronic 5-HT4 receptor activation decreases Aβ production and deposition in hAPP/PS1 mice
Neurobiology of Aging 2013cited by 54position: middledoi
Computational identification and experimental validation of microRNAs binding to the Alzheimer-related gene ADAM10
BMC Medical Genetics 2012cited by 82position: middledoi
Loss of PAFAH1B2 Reduces Amyloid-β Generation by Promoting the Degradation of Amyloid Precursor Protein C-Terminal Fragments
Journal of Neuroscience 2012cited by 29position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Stephan A. Müller · Technical University of Munich8 papers (2016–2022)Bart De Strooper · The Francis Crick Institute5 papers (2013–2021)Christian Haass · University of Regensburg5 papers (2012–2022)Peer‐Hendrik Kuhn · Sensor-Technik Wiedemann (Germany)4 papers (2012–2016)Iryna Voytyuk · University of Cambridge3 papers (2016–2021)Georg Jocher · Technical University of Munich2 papers (2022–2022)Ulrike Obermüller · University of Tübingen2 papers (2022–2022)Mathias Jucker · Yale University2 papers (2022–2022)Claus U. Pietrzik · Johannes Gutenberg University Mainz2 papers (2016–2019)Harald Steiner · Ludwig-Maximilians-Universität München2 papers (2012–2022)Wolfgang Wurst · German Center for Neurodegenerative Diseases2 papers (2012–2019)Robert Vassar · Northwestern University2 papers (2014–2021)Jörg W. Bartsch · Philipps University of Marburg2 papers (2019–2022)Marius Lambert · University of Tübingen2 papers (2022–2022)Stephan A. Kaeser · German Center for Neurodegenerative Diseases2 papers (2022–2022)Matthias Staufenbiel · University of Tübingen2 papers (2022–2022) · 2 papers (2016–2019)Christoph Becker‐Pauly · Johannes Gutenberg University Mainz2 papers (2016–2019)Kristina Endres · Johannes Gutenberg University Mainz2 papers (2012–2019)Elizabeth Hall · University of Manchester1 papers (2021–2021)