Natalie Verstraeten

The rise and future of CRISPR-based approaches for high-throughput genomics

Environmental, mechanistic and evolutionary landscape of antibiotic persistence

Ecology and evolution of antibiotic persistence

General Mechanisms Leading to Persister Formation and Awakening

HokB Monomerization and Membrane Repolarization Control Persister Awakening

GTP Binding Is Necessary for the Activation of a Toxic Mutant Isoform of the Essential GTPase ObgE

The Persistence-Inducing Toxin HokB Forms Dynamic Pores That Cause ATP Leakage

An integrative view of cell cycle control in Escherichia coli

Adaptive tuning of mutation rates allows fast response to lethal stress in Escherichia coli

New approaches to combat <i>Porphyromonas gingivalis</i> biofilms

Frequency of antibiotic application drives rapid evolutionary adaptation of Escherichia coli persistence

Molecular mechanisms and clinical implications of bacterial persistence

<i>In Vitro</i> Emergence of High Persistence upon Periodic Aminoglycoside Challenge in the ESKAPE Pathogens

Elucidation of the Mode of Action of a New Antibacterial Compound Active against Staphylococcus aureus and Pseudomonas aeruginosa

Repurposing Toremifene for Treatment of Oral Bacterial Infections

Obg and Membrane Depolarization Are Part of a Microbial Bet-Hedging Strategy that Leads to Antibiotic Tolerance

Fungal β-1,3-Glucan Increases Ofloxacin Tolerance of Escherichia coli in a Polymicrobial E. coli/Candida albicans Biofilm

Covalent immobilization of antimicrobial agents on titanium prevents<i>Staphylococcus aureus</i>and<i>Candida albicans</i>colonization and biofilm formation

Fitness trade‐offs explain low levels of persister cells in the opportunistic pathogen <i>Pseudomonas aeruginosa</i>

Derivatives of the Mouse Cathelicidin-Related Antimicrobial Peptide (CRAMP) Inhibit Fungal and Bacterial Biofilm Formation

Bacterial Obg proteins: GTPases at the nexus of protein and DNA synthesis

New-found fundamentals of bacterial persistence