Area of research
Epidemiology · Physiology
Research interest
Research interests include Adipokines, Inflammation, and Metabolic Diseases, Adipose Tissue and Metabolism, Liver Disease Diagnosis and Treatment, and Lipid metabolism and disorders.
A 5:2 intermittent fasting regimen ameliorates NASH and fibrosis and blunts HCC development via hepatic PPARα and PCK1
Beneficial effects of intermittent fasting in NASH and subsequent HCC development are executed by concerted PPARα and PCK1 action in hepatocytes
Author Correction: Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancer
GPR182 is an endothelium-specific atypical chemokine receptor that maintains hematopoietic stem cell homeostasis
Platelet GPIbα is a mediator and potential interventional target for NASH and subsequent liver cancer
Diet-dependent function of the extracellular matrix proteoglycan Lumican in obesity and glucose homeostasis
Cited4 is a sex‐biased mediator of the antidiabetic glitazone response in adipocyte progenitors
A Hepatic GAbp-AMPK Axis Links Inflammatory Signaling to Systemic Vascular Damage
A liver stress-endocrine nexus promotes metabolic integrity during dietary protein dilution
An AMP-activated protein kinase–stabilizing peptide ameliorates adipose tissue wasting in cancer cachexia in mice
Fasting‐induced liver GADD45β restrains hepatic fatty acid uptake and improves metabolic health
Mice lacking neutral amino acid transporter B0AT1 (Slc6a19) have elevated levels of FGF21 and GLP-1 and improved glycaemic control
Brown Adipose Tissue Harbors a Distinct Sub-Population of Regulatory T Cells
A Glyoxalase-1 Knockdown Does Not Have Major Short Term Effects on Energy Expenditure and Atherosclerosis in Mice
Browning of White Adipose Tissue Uncouples Glucose Uptake from Insulin Signaling
micro <scp>RNA</scp> ‐379 couples glucocorticoid hormones to dysfunctional lipid homeostasis
Hepatic transforming growth factor-β 1 stimulated clone-22 D1 controls systemic cholesterol metabolism
TSC22D4 is a molecular output of hepatic wasting metabolism
Transcriptional Cofactor TBLR1 Controls Lipid Mobilization in White Adipose Tissue