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Jorge J. Palop

Gladstone Institutes · US
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Area of research
Physiology · Cellular and Molecular Neuroscience
Research interest
Research interests include Alzheimer's disease research and treatments, Neuroscience and Neuropharmacology Research, Cholinesterase and Neurodegenerative Diseases, and Memory and Neural Mechanisms.
h-index
36
citations
15,703
works
85
NIH funding
primary concept
email

Recent publications

CRISPR screening by AAV episome-sequencing (CrAAVe-seq): a scalable cell-type-specific in vivo platform uncovers neuronal essential genes
Nature Neuroscience 2025cited by 7position: middledoi
Machine learning reveals prominent spontaneous behavioral changes and treatment efficacy in humanized and transgenic Alzheimer's disease models
Cell Reports 2024cited by 19position: lastdoi
Identifying behavioral structure from deep variational embeddings of animal motion
Communications Biology 2022cited by 158position: middledoi
Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia
Molecular Neurodegeneration 2022cited by 118position: middledoi
Additional file 13 of Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia
Figshare 2022cited by 0position: middledoi
Additional file 14 of Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia
Figshare 2022cited by 0position: middledoi
Additional file 12 of Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia
Figshare 2022cited by 0position: middledoi
Additional file 11 of Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia
Figshare 2022cited by 0position: middledoi
Additional file 15 of Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia
Figshare 2022cited by 0position: middledoi
Additional file 16 of Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia
Figshare 2022cited by 0position: middledoi
A second X chromosome contributes to resilience in a mouse model of Alzheimer’s disease
Science Translational Medicine 2020cited by 179position: middledoi
Microglial Gi-dependent dynamics regulate brain network hyperexcitability
Nature Neuroscience 2020cited by 154position: middledoi
GluN2A NMDA Receptor Enhancement Improves Brain Oscillations, Synchrony, and Cognitive Functions in Dravet Syndrome and Alzheimer’s Disease Models
Cell Reports 2020cited by 89position: lastdoi
Behavioral and neural network abnormalities in human APP transgenic mice resemble those of App knock-in mice and are modulated by familial Alzheimer’s disease mutations but not by inhibition of BACE1
Molecular Neurodegeneration 2020cited by 75position: middledoi
What electrophysiology tells us about Alzheimer's disease: a window into the synchronization and connectivity of brain neurons
Neurobiology of Aging 2019cited by 270position: middledoi
Nav1.1-Overexpressing Interneuron Transplants Restore Brain Rhythms and Cognition in a Mouse Model of Alzheimer’s Disease
Neuron 2018cited by 243position: lastdoi
Epilepsy as a Network Disorder (2): What can we learn from other network disorders such as dementia and schizophrenia, and what are the implications for translational research?
Epilepsy & Behavior 2017cited by 33position: middledoi
Nuclear pore complex remodeling by p75NTR cleavage controls TGF-β signaling and astrocyte functions
Nature Neuroscience 2015cited by 39position: middledoi
Lamin B1 mediates cell-autonomous neuropathology in a leukodystrophy mouse model
Journal of Clinical Investigation 2013cited by 79position: middledoi
Inhibitory Interneuron Deficit Links Altered Network Activity and Cognitive Dysfunction in Alzheimer Model
Cell 2012cited by 1,251position: lastdoi
Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer’s disease model
Proceedings of the National Academy of Sciences 2012cited by 644position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

Lennart Mucke · Gladstone Institutes5 papers (2012–2020)Kaitlyn Ho · Washington University in St. Louis4 papers (2012–2020)Laure Verret · Centre National de la Recherche Scientifique4 papers (2012–2018)Gui‐Qiu Yu · Gladstone Institutes3 papers (2012–2020)Katerina Akassoglou · Gladstone Institutes3 papers (2015–2024)Keran Ma · First Affiliated Hospital of Zhengzhou University3 papers (2018–2025)Nino Devidze · Gladstone Institutes3 papers (2012–2020)Stephanie R. Miller · Gladstone Institutes2 papers (2022–2024)Jae Kyu Ryu · Gladstone Institutes2 papers (2015–2020)Inma Cobos · Stanford Medicine2 papers (2012–2018)Victoria A. Rafalski · Gladstone Institutes2 papers (2015–2020) · 2 papers (2022–2024)Reuben Thomas · Gladstone Institutes2 papers (2020–2024)Md. Abdullah Saeed Khan · Bolan University of Medical and Health Sciences2 papers (2015–2018)Andrew S. Mendiola · Gladstone Institutes2 papers (2020–2024)Zhaoqi Yan · Jiangxi University of Traditional Chinese Medicine2 papers (2020–2024)Mark H. Ellisman · NCR (United States)2 papers (2015–2020) · 2 papers (2022–2024) · 2 papers (2022–2024)Pascal E. Sanchez · Denali Therapeutics (United States)2 papers (2012–2020)
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