Area of research
Genetics · Hematology
Research interest
Research focused on FOXM1 and Cancer research, with related work in Malaria, Disease, Hemolysis. Notable publications include 'Erythrocytic ferroportin reduces intracellular iron accumulation, hemolysis, and malaria risk', 'The impact of delayed treatment of uncomplicated P. falciparum malaria on progression to severe malaria: A systematic review and a pooled multicentre individual-patient...', and 'Gain-of-function EGLN1 prolyl hydroxylase (PHD2 D4E:C127S) in combination with EPAS1 (HIF-2α) polymorphism lowers hemoglobin concentration in Tibetan highlanders'.
Increased prevalence of clonal hematopoiesis in children with sickle cell disease
Alkaline Phosphatase As a Marker for Acute Complications in Sickle Cell Disease
Novel FOXM1 inhibitor identified via gene network analysis induces autophagic FOXM1 degradation to overcome chemoresistance of human cancer cells
The impact of delayed treatment of uncomplicated P. falciparum malaria on progression to severe malaria: A systematic review and a pooled multicentre individual-patient meta-analysis
Clinical, laboratory, and genetic risk factors for thrombosis in sickle cell disease
The <scp><i>CYB5R3</i><sup>c</sup></scp><sup>.<scp>350C</scp>>G</sup> and <scp><i>G6PD</i></scp> A alleles modify severity of anemia in malaria and sickle cell disease
Phlebotomy-Induced Iron Deficiency Increases the Expression of Prothrombotic Genes
Erythrocytic ferroportin reduces intracellular iron accumulation, hemolysis, and malaria risk
Honokiol is a FOXM1 antagonist
HMOX1 and acute kidney injury in sickle cell anemia
Risk factors for vitamin D deficiency in sickle cell disease
Hemolysis and hemolysis‐related complications in females vs. males with sickle cell disease
Fixed low‐dose hydroxyurea for the treatment of adults with sickle cell anemia in<scp>N</scp>igeria
Gain-of-function EGLN1 prolyl hydroxylase (PHD2 D4E:C127S) in combination with EPAS1 (HIF-2α) polymorphism lowers hemoglobin concentration in Tibetan highlanders
Prospective study of thrombosis and thrombospondin-1 expression in Chuvash polycythemia
Association of circulating transcriptomic profiles with mortality in sickle cell disease
<i>APOL1</i> , α-thalassemia, and <i>BCL11A</i> variants as a genetic risk profile for progression of chronic kidney disease in sickle cell anemia
Hyperfiltration is associated with the development of microalbuminuria in patients with sickle cell anemia