Area of research
Physiology · Molecular Biology
Research interest
Research interests include Adipose Tissue and Metabolism, Metabolism, Diabetes, and Cancer, Cellular transport and secretion, and Pancreatic function and diabetes.
Cold exposure stimulates cross-tissue metabolic rewiring to fuel glucose-dependent thermogenesis in brown adipose tissue
Deep Proteome Profiling of White Adipose Tissue Reveals Marked Conservation and Distinct Features Between Different Anatomical Depots
Systems-level analysis of insulin action in mouse strains provides insight into tissue- and pathway-specific interactions that drive insulin resistance
The aetiology and molecular landscape of insulin resistance
Proteomics analysis of adipose depots after intermittent fasting reveals visceral fat preservation mechanisms
Phosphoproteomics reveals conserved exercise‐stimulated signaling and AMPK regulation of store‐operated calcium entry
Mitochondrial oxidative stress causes insulin resistance without disrupting oxidative phosphorylation
High dietary fat and sucrose result in an extensive and time-dependent deterioration in health of multiple physiological systems in mice
A novel Rab10-EHBP1-EHD2 complex essential for the autophagic engulfment of lipid droplets
mTORC1 Is a Major Regulatory Node in the FGF21 Signaling Network in Adipocytes
Global Phosphoproteomic Analysis of Human Skeletal Muscle Reveals a Network of Exercise-Regulated Kinases and AMPK Substrates
Selective Insulin Resistance in Adipocytes
Kinome Screen Identifies PFKFB3 and Glucose Metabolism as Important Regulators of the Insulin/Insulin-like Growth Factor (IGF)-1 Signaling Pathway
Proteomic Analysis of GLUT4 Storage Vesicles Reveals Tumor Suppressor Candidate 5 (TUSC5) as a Novel Regulator of Insulin Action in Adipocytes
The RabGAP TBC1D1 Plays a Central Role in Exercise-Regulated Glucose Metabolism in Skeletal Muscle
Dynamic Adipocyte Phosphoproteome Reveals that Akt Directly Regulates mTORC2
Impaired Akt phosphorylation in insulin-resistant human muscle is accompanied by selective and heterogeneous downstream defects