Area of research
Psychiatry and Mental health · Ophthalmology
Research interest
Research focused on Candida albicans and Microbiology, with related work in Pneumolysin, Miller, Syk. Notable publications include 'Neutrophil IL-1β Processing Induced by Pneumolysin Is Mediated by the NLRP3/ASC Inflammasome and Caspase-1 Activation and Is Dependent on K+ Efflux', 'Infectious corneal ulceration: a proposal for neglected tropical disease status', and 'Tyrosine phosphatase SHP-2 mediates C-type lectin receptor–induced activation of the kinase Syk and anti-fungal TH17 responses'.
Host-specific adaptation in <i>Fusarium oxysporum</i> correlates with distinct accessory chromosome content in human and plant pathogenic strains
Comparing the Efficacy and Safety of Galcanezumab Versus Rimegepant for Prevention of Episodic Migraine: Results from a Randomized, Controlled Clinical Trial
Immunity to pathogenic fungi in the eye
Control of β-glucan exposure by the endo-1,3-glucanase Eng1 in Candida albicans modulates virulence
American Academy of Optometry Microbial Keratitis Think Tank
The genome of opportunistic fungal pathogen Fusarium oxysporum carries a unique set of lineage-specific chromosomes
Assessment of immunogenicity from galcanezumab phase 3 trials in patients with episodic or chronic migraine
Infectious corneal ulceration: a proposal for neglected tropical disease status
EphA2 Is a Neutrophil Receptor for Candida albicans that Stimulates Antifungal Activity during Oropharyngeal Infection
Linked Toll-Like Receptor Triagonists Stimulate Distinct, Combination-Dependent Innate Immune Responses
Tuning Subunit Vaccines with Novel TLR Triagonist Adjuvants to Generate Protective Immune Responses against <i>Coxiella burnetii</i>
Immunogenicity Assessment from Phase 3 Galcanezumab Trials in Patients with Episodic or Chronic Migraine (P1.10-017)
Neutrophil IL-1β Processing Induced by Pneumolysin Is Mediated by the NLRP3/ASC Inflammasome and Caspase-1 Activation and Is Dependent on K+ Efflux
Tyrosine phosphatase SHP-2 mediates C-type lectin receptor–induced activation of the kinase Syk and anti-fungal TH17 responses