Area of research
Immunology · Molecular Biology
Research interest
Research focused on Cell biology and Inflammation, with related work in Immunology, Autoimmunity, Tuft. Notable publications include 'TLR signaling adapter BCAP regulates inflammatory to reparatory macrophage transition by promoting histone lactylation', 'T cells instruct myeloid cells to produce inflammasome-independent IL-1β and cause autoimmunity', and 'Injury-induced pulmonary tuft cells are heterogenous, arise independent of key Type 2 cytokines, and are dispensable for dysplastic repair'.
Injury-induced pulmonary tuft cells are heterogenous, arise independent of key Type 2 cytokines, and are dispensable for dysplastic repair
Effector memory CD4<sup>+</sup> T cells induce damaging innate inflammation and autoimmune pathology by engaging CD40 and TNFR on myeloid cells.
TLR signaling adapter BCAP regulates inflammatory to reparatory macrophage transition by promoting histone lactylation
T cells instruct myeloid cells to produce inflammasome-independent IL-1β and cause autoimmunity