Area of research
Physiology · Materials Chemistry
Research interest
Research interests include Alzheimer's disease research and treatments, Enzyme Structure and Function, Protein Structure and Dynamics, and Autophagy in Disease and Therapy.
Zipper-interacting Protein Kinase Modulates Gene Expression Linked to Synaptic and Neuronal Processes after Traumatic Brain Injury.
Zipper-interacting protein kinase mediates neuronal cell death and cognitive dysfunction in traumatic brain injury via regulating DEDD.
Multifaceted role of zipper-interacting protein kinase beyond cell death: Implication of ZIPK dysregulation in neuronal and vascular injuries.
Death-associated protein kinase 1 as a therapeutic target for Alzheimer's disease.
Activation of transient receptor potential vanilloid 1 ameliorates tau accumulation-induced synaptic damage and cognitive dysfunction via autophagy enhancement.
Quantitative Proteomic and Phosphoproteomic Analyses Reveal a Role of Death-Associated Protein Kinase 1 in Regulating Hippocampal Synapse.
Death-associated protein kinase 1 phosphorylates MDM2 and inhibits its protein stability and function.
Mitochondria-Targeted Fluorescent Nanoparticles with Large Stokes Shift for Long-Term BioImaging
Development and evolution of human glutaminyl cyclase inhibitors (QCIs): an alternative promising approach for disease-modifying treatment of Alzheimer's disease.
Differentiation of subnucleus-sized oligomers and nucleation-competent assemblies of the Aβ peptide.
Death-associated protein kinase 1 mediates Aβ42 aggregation-induced neuronal apoptosis and tau dysregulation in Alzheimer's disease.
Additional file 2 of iTRAQ-based proteomic analysis of Deinococcus radiodurans in response to 12C6+ heavy ion irradiation
Antimony-induced astrocyte activation via mitogen-activated protein kinase activation-dependent CREB phosphorylation
An essential role of the autophagy activating kinase ULK1 in snRNP biogenesis.
Quantitative proteomics reveals that dormancy-related proteins mediate the attenuation in mycobacterium strains
Recognition and Removal of Amyloid‐β by a Heteromultivalent Macrocyclic Coassembly: A Potential Strategy for the Treatment of Alzheimer's Disease
The Genetics of Alzheimer's Disease in the Chinese Population.
Farnesylation of human guanylate-binding protein 1 as safety mechanism preventing structural rearrangements and uninduced dimerization.
Phosphorylation Signaling in APP Processing in Alzheimer's Disease.
Molecular adaptations of NADP-malic enzyme for its function in C<sub>4</sub> photosynthesis in grasses.
Toward the Mode of Action of the Clinical Stage All-d-Enantiomeric Peptide RD2 on Aβ42 Aggregation.
Solution-Based Determination of Dissociation Constants for the Binding of Aβ42 to Antibodies.
Autophagy Is a Cell Self-Protective Mechanism Against Arsenic-Induced Cell Transformation