Area of research
Molecular Biology · Cellular and Molecular Neuroscience
Research interest
Research focused on Corticogenesis and Neural stem cell, with related work in Neuroscience, Neuroprotection, XBP1. Notable publications include 'XBP1 (X-Box–Binding Protein-1)–Dependent O-GlcNAcylation Is Neuroprotective in Ischemic Stroke in Young Mice and Its Impairment in Aged Mice Is Rescued by Thiamet-G', 'O-GlcNAcylation promotes cerebellum development and medulloblastoma oncogenesis via SHH signaling', and 'Loss of O-GlcNAc transferase in neural stem cells impairs corticogenesis'.
The genetic landscape of early‐onset Alzheimer's disease in China
Active Micro-Nano-Collaborative Bioelectronic Device for Advanced Electrophysiological Recording
Overexpression of ESYT3 improves radioimmune responses through activating cGAS-STING pathway in lung adenocarcinoma
O-GlcNAcylation Is Required for the Survival of Cerebellar Purkinje Cells by Inhibiting ROS Generation
Proteome Profiling of Serum Exosomes from Newborns with Lung Injury after Perinatal Asphyxia
O-GlcNAcylation promotes cerebellum development and medulloblastoma oncogenesis via SHH signaling
Scalable and Robust Hollow Nanopillar Electrode for Enhanced Intracellular Action Potential Recording
CaMKIIα Signaling Is Required for the Neuroprotective Effects of Dl-3-n-Butylphthalide in Alzheimer’s Disease
Rack1 is essential for corticogenesis by preventing p21-dependent senescence in neural stem cells
Loss of O-GlcNAcylation on MeCP2 at Threonine 203 Leads to Neurodevelopmental Disorders
Loss of O-GlcNAc transferase in neural stem cells impairs corticogenesis
Transferrin receptor 1 plays an important role in muscle development and denervation-induced muscular atrophy
Rack1 Controls Parallel Fiber–Purkinje Cell Synaptogenesis and Synaptic Transmission
XBP1 (X-Box–Binding Protein-1)–Dependent O-GlcNAcylation Is Neuroprotective in Ischemic Stroke in Young Mice and Its Impairment in Aged Mice Is Rescued by Thiamet-G
Elevated Plasma Angiogenesis Factors in Alzheimer's Disease