Area of research
Pulmonary and Respiratory Medicine · Physiology
Research interest
Research focused on Pulmonary hypertension and Pneumolysin, with related work in Inflammation, Glycolysis, NOX4. Notable publications include 'PFKFB3-mediated endothelial glycolysis promotes pulmonary hypertension', 'NADPH Oxidase 4 Is Expressed in Pulmonary Artery Adventitia and Contributes to Hypertensive Vascular Remodeling', and 'Role of C/EBP homologous protein and endoplasmic reticulum stress in asthma exacerbation by regulating the IL-4/signal transducer and activator of transcription 6/transcription...'.
PDZ-Binding Kinase, a Novel Regulator of Vascular Remodeling in Pulmonary Arterial Hypertension
Clinicopathological Correlation of Chronic Thromboembolic Pulmonary Hypertension: A Retrospective Study
PDZ‐Binding Kinase Drives Pulmonary Artery Smooth Muscle Proliferation and Vascular Remodeling in Pulmonary Arterial Hypertension
Deficiency of Myeloid Pfkfb3 Protects Mice From Lung Edema and Cardiac Dysfunction in LPS-Induced Endotoxemia
Niacin Attenuates Pulmonary Hypertension Through H-PGDS in Macrophages
Dual Role of Hydrogen Peroxide as an Oxidant in Pneumococcal Pneumonia
PFKFB3-mediated endothelial glycolysis promotes pulmonary hypertension
Galectin-3 is expressed in vascular smooth muscle cells and promotes pulmonary hypertension through changes in proliferation, apoptosis, and fibrosis
Polygonum aviculare L. extract and quercetin attenuate contraction in airway smooth muscle
Role of C/EBP homologous protein and endoplasmic reticulum stress in asthma exacerbation by regulating the IL-4/signal transducer and activator of transcription 6/transcription factor EC/IL-4 receptor α positive feedback loop in M2 macrophages
Epithelial Sodium Channel-α Mediates the Protective Effect of the TNF-Derived TIP Peptide in Pneumolysin-Induced Endothelial Barrier Dysfunction
NADPH Oxidase 4 Is Expressed in Pulmonary Artery Adventitia and Contributes to Hypertensive Vascular Remodeling
Endothelin-1 Induces a Glycolytic Switch in Pulmonary Arterial Endothelial Cells via the Mitochondrial Translocation of Endothelial Nitric Oxide Synthase