Area of research
Cellular and Molecular Neuroscience · Physiology
Research interest
Research interests include Neuroscience and Neuropharmacology Research, Alzheimer's disease research and treatments, Neuroinflammation and Neurodegeneration Mechanisms, and Memory and Neural Mechanisms.
Prognostic value of nighttime blood pressure in patients with chronic kidney disease
Macrophage iron dyshomeostasis promotes aging‐related renal fibrosis
Impact of lactate/albumin ratio on prognostic outcomes in patients with concomitant heart failure and chronic kidney disease
Disruption of the IL-33-ST2-AKT signaling axis impairs neurodevelopment by inhibiting microglial metabolic adaptation and phagocytic function
Transcriptional Circuitry of NKX2-1 and SOX1 Defines an Unrecognized Lineage Subtype of Small-Cell Lung Cancer
Expert consensus on resection of chest wall tumors and chest wall reconstruction
Neuronal Loss of the Glutamate Transporter GLT-1 Promotes Excitotoxic Injury in the Hippocampus
A mechanistic hypothesis for the impairment of synaptic plasticity by soluble Aβ oligomers from Alzheimer’s brain
Environmental enrichment prevents Aβ oligomer-induced synaptic dysfunction through mirna-132 and hdac3 signaling pathways
Metabolic profiles of serum samples from ground glass opacity represent potential diagnostic biomarkers for lung cancer
Crucial Roles for SIRT2 and AMPA Receptor Acetylation in Synaptic Plasticity and Memory
Evening versus morning dosing regimen drug therapy for chronic kidney disease patients with hypertension in blood pressure patterns: a systematic review and meta‐analysis
Complement and microglia mediate early synapse loss in Alzheimer mouse models
Large Soluble Oligomers of Amyloid β-Protein from Alzheimer Brain Are Far Less Neuroactive Than the Smaller Oligomers to Which They Dissociate
Building neurophenomics in zebrafish: Effects of prior testing stress and test batteries
Complement<i>C3</i>-Deficient Mice Fail to Display Age-Related Hippocampal Decline
Soluble Aβ oligomers impair hippocampal LTP by disrupting glutamatergic/GABAergic balance
Rosiglitazone Prevents Amyloid-β Oligomer-Induced Impairment of Synapse Formation and Plasticity via Increasing Dendrite and Spine Mitochondrial Number