James J. Peters VA Medical Center · US
Area of research
Physiology · Pharmacology
Research interest
Research interests include Alzheimer's disease research and treatments, Cholinesterase and Neurodegenerative Diseases, Dementia and Cognitive Impairment Research, and Traumatic Brain Injury Research.
The contribution of amyloid deposition in the aortic valve to calcification and aortic stenosis
ApoE Alzheimer’s Disease Aβ-amyloid plaque morphology varies according to APOE isotype
Alzheimer’s Disease A<i>β</i>-Amyloid Plaque Morphology Varies According to <i>APOE</i> Isotype
Altered succinylation of mitochondrial proteins, APP and tau in Alzheimer’s disease
Microglial <i>INPP5D</i> limits plaque formation and glial reactivity in the PSAPP mouse model of Alzheimer's disease
Interpretable deep learning of myelin histopathology in age-related cognitive impairment
Differential Vulnerability of Hippocampal Subfields in Primary Age-Related Tauopathy and Chronic Traumatic Encephalopathy
Molecular subtyping of Alzheimer’s disease using RNA sequencing data reveals novel mechanisms and targets
Low-level blast exposure induces chronic vascular remodeling, perivascular astrocytic degeneration and vascular-associated neuroinflammation
Cognitive impairment and World Trade Centre-related exposures
Genome-wide association study and functional validation implicates JADE1 in tauopathy
Laterality and region-specific tau phosphorylation correlate with PTSD-related behavioral traits in rats exposed to repetitive low-level blast
Transcranial Laser Therapy Does Not Improve Cognitive and Post-Traumatic Stress Disorder–Related Behavioral Traits in Rats Exposed to Repetitive Low-Level Blast Injury
Genome-wide association study and functional validation implicates JADE1 in tauopathy
Transformative Network Modeling of Multi-omics Data Reveals Detailed Circuits, Key Regulators, and Potential Therapeutics for Alzheimer’s Disease
Multiscale causal networks identify VGF as a key regulator of Alzheimer’s disease
VGF-derived peptide TLQP-21 modulates microglial function through C3aR1 signaling pathways and reduces neuropathology in 5xFAD mice
Brain and blood biomarkers of tauopathy and neuronal injury in humans and rats with neurobehavioral syndromes following blast exposure
Low-level blast exposure disrupts gliovascular and neurovascular connections and induces a chronic vascular pathology in rat brain
Molecular Networks and Key Regulators of the Dysregulated Neuronal System in Alzheimer’s Disease
Unexpected systemic phenotypes result from focal combined deficiencies of forebrain insulin receptor/IGF-1 receptor signaling
Brain insulin resistance in type 2 diabetes and Alzheimer disease: concepts and conundrums
Multiscale Analysis of Independent Alzheimer’s Cohorts Finds Disruption of Molecular, Genetic, and Clinical Networks by Human Herpesvirus
The Mount Sinai cohort of large-scale genomic, transcriptomic and proteomic data in Alzheimer's disease
Blast-induced "PTSD": Evidence from an animal model
Directed Differentiation of Human Pluripotent Stem Cells to Microglia
Antemortem biomarker support for a diagnosis of clinically probable chronic traumatic encephalopathy
Integrative network analysis of nineteen brain regions identifies molecular signatures and networks underlying selective regional vulnerability to Alzheimer’s disease
Cerebral [18 F]T807/AV1451 retention pattern in clinically probable CTE resembles pathognomonic distribution of CTE tauopathy
Effective anti-Alzheimer Aβ therapy involves depletion of specific Aβ oligomer subtypes