Area of research
Oceanography · Industrial and Manufacturing Engineering
Research interest
Research interests include Marine and coastal ecosystems, Water Quality Monitoring and Analysis, Water Quality Monitoring Technologies, and Inflammasome and immune disorders.
Molecular basis for potent B cell responses to antigen displayed on particles of viral size
NR4A nuclear receptors in T and B lymphocytes: Gatekeepers of immune tolerance*
NR4A family members regulate T cell tolerance to preserve immune homeostasis and suppress autoimmunity
ATP-competitive partial antagonists of the IRE1α RNase segregate outputs of the UPR
NR4A nuclear receptors restrain B cell responses to antigen when second signals are absent or limiting
Alternative splicing regulates stochastic NLRP3 activity
Nur77 Links Chronic Antigen Stimulation to B Cell Tolerance by Restricting the Survival of Self-Reactive B Cells in the Periphery
Optimal Development of Mature B Cells Requires Recognition of Endogenous Antigens
IgM and IgD B cell receptors differentially respond to endogenous antigens and control B cell fate
<i>EPCAM</i>mutation update: Variants associated with congenital tufting enteropathy and Lynch syndrome
IL-2 Modulates the TCR Signaling Threshold for CD8 but Not CD4 T Cell Proliferation on a Single-Cell Level
Nur77 Is Upregulated in B-1a Cells by Chronic Self-Antigen Stimulation and Limits Generation of Natural IgM Plasma Cells
An extracatalytic function of CD45 in B cells is mediated by CD22
ORMDL3 Transgenic Mice Have Increased Airway Remodeling and Airway Responsiveness Characteristic of Asthma
A sharp T-cell antigen receptor signaling threshold for T-cell proliferation
Divergence of IL-1, IL-18, and cell death in NLRP3 inflammasomopathies
Absence of cell-surface EpCAM in congenital tufting enteropathy
ORMDL3 is an inducible lung epithelial gene regulating metalloproteases, chemokines, OAS, and ATF6
Constitutively Activated NLRP3 Inflammasome Causes Inflammation and Abnormal Skeletal Development in Mice
Cutting Edge: IL-6 Is a Marker of Inflammation with No Direct Role in Inflammasome-Mediated Mouse Models