Area of research
Molecular Biology · Pathology and Forensic Medicine
Research interest
Research focused on Intestinal permeability and Inflammation, with related work in Gut flora, Neuroinflammation, Parkinson's disease. Notable publications include 'Role of TLR4 in the gut-brain axis in Parkinson’s disease: a translational study from men to mice', 'Chronic stress-induced gut dysfunction exacerbates Parkinson's disease phenotype and pathology in a rotenone-induced mouse model of Parkinson's disease', and 'Progression of intestinal permeability changes and alpha‐synuclein expression in a mouse model of Parkinson's disease'.
Distinct intestinal microbial signatures linked to accelerated systemic and intestinal biological aging
The Bidirectional Effects of Periodontal Disease and Oral Dysbiosis on Gut Inflammation in Inflammatory Bowel Disease
An open label, non-randomized study assessing a prebiotic fiber intervention in a small cohort of Parkinson’s disease participants
Microglia, inflammation and gut microbiota responses in a progressive monkey model of Parkinson's disease: A case series
Abnormal food timing and predisposition to weight gain: Role of barrier dysfunction and microbiota
Abnormal Eating Patterns Cause Circadian Disruption and Promote Alcohol-Associated Colon Carcinogenesis
Role of TLR4 in the gut-brain axis in Parkinson’s disease: a translational study from men to mice
Chronic stress-induced gut dysfunction exacerbates Parkinson's disease phenotype and pathology in a rotenone-induced mouse model of Parkinson's disease
Dietary Fiber Treatment Corrects the Composition of Gut Microbiota, Promotes SCFA Production, and Suppresses Colon Carcinogenesis
Alcohol Feeding in Mice Promotes Colonic Hyperpermeability and Changes in Colonic Organoid Stem Cell Fate
Simultaneous gas-chromatographic urinary measurement of sugar probes to assess intestinal permeability: Use of time course analysis to optimize its use to assess regional gut permeability
Progression of intestinal permeability changes and alpha‐synuclein expression in a mouse model of Parkinson's disease
Disruption of the Circadian Clock in Mice Increases Intestinal Permeability and Promotes Alcohol-Induced Hepatic Pathology and Inflammation
Role for intestinal CYP2E1 in alcohol-induced circadian gene-mediated intestinal hyperpermeability