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Qing Shi

Fudan University · CN
Area of research
Public Health, Environmental and Occupational Health · Molecular Biology
Research interest
Research focused on Cancer research and Ubiquitin ligase, with related work in Prostate cancer, Ubiquitin, DNA methylation. Notable publications include 'Prostate Cancer-associated SPOP mutations enhance cancer cell survival and docetaxel resistance by upregulating Caprin1-dependent stress granule assembly', 'SPOP mutation induces DNA methylation via stabilizing GLP/G9a', and 'SPOP promotes ATF2 ubiquitination and degradation to suppress prostate cancer progression'.
h-index
citations
510
works
11
NIH funding
primary concept
email

Recent publications

Exploring the therapeutic potential of natural compounds for Alzheimer's disease: Mechanisms of action and pharmacological properties
Biomedicine & Pharmacotherapy 2023cited by 40position: lastdoi
Multifunctional Gomisin B enhances cognitive function in APP/PS1 transgenic mice by regulating Aβ clearance and neuronal apoptosis
Biomedicine & Pharmacotherapy 2023cited by 10position: middledoi
SPOP mutations promote tumor immune escape in endometrial cancer via the IRF1–PD-L1 axis
Cell Death and Differentiation 2022cited by 50position: middledoi
A nomogram for predicting prognosis of multiple myeloma patients based on a ubiquitin-proteasome gene signature
Aging 2022cited by 5position: middledoi
Development and Internal Validation of a Model Predicting the Risk of Recurrent Stroke for Middle-Aged and Elderly Patients: A Retrospective Cohort Study
World Neurosurgery 2022cited by 0position: middledoi
SPOP mutation induces DNA methylation via stabilizing GLP/G9a
Nature Communications 2021cited by 69position: middledoi
SPOP mutation induces replication over-firing by impairing Geminin ubiquitination and triggers replication catastrophe upon ATR inhibition
Nature Communications 2021cited by 32position: middledoi
Prostate Cancer-associated SPOP mutations enhance cancer cell survival and docetaxel resistance by upregulating Caprin1-dependent stress granule assembly
Molecular Cancer 2019cited by 130position: firstdoi
CRL3–SPOP ubiquitin ligase complex suppresses the growth of diffuse large B-cell lymphoma by negatively regulating the MyD88/NF-κB signaling
Leukemia 2019cited by 58position: middledoi
SPOP promotes ATF2 ubiquitination and degradation to suppress prostate cancer progression
Journal of Experimental & Clinical Cancer Research 2018cited by 64position: middledoi
ARRDC1 and ARRDC3 act as tumor suppressors in renal cell carcinoma by facilitating YAP1 degradation.
PubMed 2018cited by 52position: middle

Grants

No grants ingested yet.

Frequent collaborators

Chenji Wang · Fudan University6 papers (2018–2022)Kun Gao · Fudan University5 papers (2018–2022)Haojie Huang · Indiana University School of Medicine3 papers (2019–2021)Pingzhao Zhang · Fudan University3 papers (2018–2021)Yao Li · Fudan University3 papers (2018–2021)Jian Ma · Shanghai Medical College of Fudan University3 papers (2018–2021)Jinman Liu · Guangzhou University of Chinese Medicine2 papers (2023–2023)Yuqian Yan · Nanchang University2 papers (2021–2021)Kai Feng · Fudan University2 papers (2018–2019)Kun Chang · Fudan University2 papers (2018–2019)Liguo Wang · University of Pennsylvania2 papers (2021–2021)Yuzhuo Wang · China Agricultural University2 papers (2021–2021)Huiru Sun · Fudan University1 papers (2019–2019)Yan Ding · The First Affiliated Hospital, Sun Yat-sen University1 papers (2022–2022)Shimin Zhao · Fudan University1 papers (2021–2021)Zhiwang Song · Nanchang University1 papers (2022–2022)Yunqian Pan · Indiana University School of Medicine1 papers (2021–2021)Yang Bai · The Affiliated Yongchuan Hospital of Chongqing Medical University1 papers (2019–2019) · 1 papers (2021–2021)Ren Mo · Shanghai Medical College of Fudan University1 papers (2019–2019)