Area of research
Molecular Biology · Physiology
Research interest
Research interests include Biology, Skeletal muscle, Mitochondrion, Cell biology, Sarcopenia, and Mitophagy.
C16ORF70/MYTHO promotes healthy aging in C.elegans and prevents cellular senescence in mammals
Intracellular to Interorgan Mitochondrial Communication in Striated Muscle in Health and Disease
Implications of mitochondrial fusion and fission in skeletal muscle mass and health
Mechanisms of muscle atrophy and hypertrophy: implications in health and disease
The connection between the dynamic remodeling of the mitochondrial network and the regulation of muscle mass
The Interplay between Mitochondrial Morphology and Myomitokines in Aging Sarcopenia
DRP1-mediated mitochondrial shape controls calcium homeostasis and muscle mass
FGF21 as Modulator of Metabolism in Health and Disease
Fibroblast growth factor 21 controls mitophagy and muscle mass
Inhibition of the Fission Machinery Mitigates OPA1 Impairment in Adult Skeletal Muscles
Skeletal muscle mTORC1 regulates neuromuscular junction stability
Transcriptional programming of lipid and amino acid metabolism by the skeletal muscle circadian clock
Age-Associated Loss of OPA1 in Muscle Impacts Muscle Mass, Metabolic Homeostasis, Systemic Inflammation, and Epithelial Senescence
In mammalian skeletal muscle, phosphorylation of TOMM22 by protein kinase CSNK2/CK2 controls mitophagy
Muscle Expression of <i> SOD1 <sup>G93A</sup> </i> Triggers the Dismantlement of Neuromuscular Junction <i>via</i> PKC-Theta
Mitochondrial Quality Control and Muscle Mass Maintenance
Transcription Factor EB Controls Metabolic Flexibility during Exercise
Glycolytic-to-oxidative fiber-type switch and mTOR signaling activation are early-onset features of SBMA muscle modified by high-fat diet
Physical exercise in aging human skeletal muscle increases mitochondrial calcium uniporter expression levels and affects mitochondria dynamics
Regulation of autophagy and the ubiquitin–proteasome system by the FoxO transcriptional network during muscle atrophy
The Opa1-Dependent Mitochondrial Cristae Remodeling Pathway Controls Atrophic, Apoptotic, and Ischemic Tissue Damage
Lifelong Physical Exercise Delays Age-Associated Skeletal Muscle Decline
Electrical Stimulation Counteracts Muscle Decline in Seniors
Signalling pathways regulating muscle mass in ageing skeletal muscle. The role of the IGF1-Akt-mTOR-FoxO pathway
Mitochondrial biogenesis and fragmentation as regulators of protein degradation in striated muscles