Area of research
Infectious Diseases · Biochemistry
Research interest
Research interests include Chemistry, Apoptosis, Cardioprotection, Spermine, Mycobacterium tuberculosis, and Biology.
Exogenous spermine attenuates myocardial fibrosis in diabetic cardiomyopathy by inhibiting endoplasmic reticulum stress and the canonical Wnt signaling pathway
Rufomycin Targets ClpC1 Proteolysis in Mycobacterium tuberculosis and M. abscessus
H2S restores the cardioprotective effects of ischemic post-conditioning by upregulating HB-EGF/EGFR signaling
Calcium‑sensing receptor promotes high glucose‑induced myocardial fibrosis via upregulation of the TGF‑β1/Smads pathway in cardiac fibroblasts
Calhex<sub>231</sub> Alleviates High Glucose-Induced Myocardial Fibrosis <i>via</i> Inhibiting Itch-Ubiquitin Proteasome Pathway <i>in Vitro</i>
[Exogenous spermine alleviates myocardial injury induced by hyperglycemia by inhibiting golgi stress].
[Changes of myocardial function in diabetic rats and its mechanism].
Pyrazole and imidazo[1,2-b]pyrazole Derivatives as New Potential Antituberculosis Agents
EXOGENOUS H2S CONTRIBUTES TO RECOVERY OF ISCHEMIC POST-CONDITIONING-INDUCED CARDIOPROTECTION VIA INCREASE OF AUTOPHAGY IN THE AGED HEARTS
[Protective effects of exogenous H<sub>2</sub>S to the recovery of hypoxia post-conditioning on aged H9C2 cells and the underling mechanisms].
Exogenous H2S restores ischemic post-conditioning-induced cardioprotection through inhibiting endoplasmic reticulum stress in the aged cardiomyocytes
Discovery of Novel Oral Protein Synthesis Inhibitors of Mycobacterium tuberculosis That Target Leucyl-tRNA Synthetase
Involvement of exogenous H2S in recovery of cardioprotection from ischemic post-conditioning via increase of autophagy in the aged hearts
Exogenous H2S contributes to recovery of ischemic post-conditioning-induced cardioprotection by decrease of ROS level via down-regulation of NF-κB and JAK2-STAT3 pathways in the aging cardiomyocytes
Spermine inhibits Endoplasmic Reticulum Stress - induced Apoptosis: a New Strategy to Prevent Cardiomyocyte Apoptosis
Exogenous spermine inhibits hypoxia/ischemia-induced myocardial apoptosis via regulation of mitochondrial permeability transition pore and associated pathways
Mediation of exogenous hydrogen sulfide in recovery of ischemic post-conditioning-induced cardioprotection via down-regulating oxidative stress and up-regulating PI3K/Akt/GSK-3β pathway in isolated aging rat hearts
Exogenous spermine inhibits the proliferation of human pulmonary artery smooth muscle cells caused by chemically-induced hypoxia via the suppression of the ERK1/2- and PI3K/AKT-associated pathways
Exogenous spermine ameliorates high glucose-induced cardiomyocytic apoptosis via decreasing reactive oxygen species accumulation through inhibiting p38/JNK and JAK2 pathways.
PubMed 2015cited by 20position: middle
glpx Gene in Mycobacterium tuberculosis Is Required for In Vitro Gluconeogenic Growth and In Vivo Survival
Dopamine D2 receptors contribute to cardioprotection of ischemic post-conditioning via activating autophagy in isolated rat hearts
Targeting Pin1 Protects Mouse Cardiomyocytes from High‐Dose Alcohol‐Induced Apoptosis
Novel Insights into the Mechanism of Inhibition of MmpL3, a Target of Multiple Pharmacophores in Mycobacterium tuberculosis
High-dose alcohol induces reactive oxygen species-mediated apoptosis via PKC-β/p66Shc in mouse primary cardiomyocytes
Cytotoxic Constituents from <i>Lobaria scrobiculata</i> and a Comparison of Two Bioassays for Their Evaluation
Gene expression profile analysis of rat cerebellum under acute alcohol intoxication
Chlorinated Coumarins from the Polypore Mushroom <i>Fomitopsis officinalis</i> and Their Activity against <i>Mycobacterium tuberculosis</i>
Inverse Correlation Between Circulating Endothelial Progenitor Cells With CD34+CD133+ and the Severity of Coronary Atherosclerosis Assessed by Syntax Score
The Oxidation-sensing Regulator (MosR) Is a New Redox-dependent Transcription Factor in Mycobacterium tuberculosis
Unbiased evaluation of bioactive secondary metabolites in complex matrices