Area of research
Biochemistry · Clinical Biochemistry
Research interest
Research focused on Cardioprotection and Mitochondrion, with related work in Protein kinase B, Sodium hydrosulfide, Calcium-sensing receptor. Notable publications include 'Hydrogen sulfide (H 2 S) metabolism in mitochondria and its regulatory role in energy production', 'Oxygen-sensitive mitochondrial accumulation of cystathionine β-synthase mediated by Lon protease', and 'Involvement of exogenous H2S in recovery of cardioprotection from ischemic post-conditioning via increase of autophagy in the aged hearts'.
Localized delivery and retention of hydrogen sulfide causing regional lipid accumulation in mouse adipose tissues in vivo
Deficiency of cystathionine gamma-lyase promotes aortic elastolysis and medial degeneration in aged mice
Hydrogen Sulfide Promotes Adipocyte Differentiation, Hyperplasia, and Hypertrophy
Cystathionine gamma‐lyase/H <sub>2</sub> S signaling facilitates myogenesis under aging and injury condition
Effect of hydrogen sulfide on glycolysis‐based energy production in mouse erythrocytes
Interaction among estrogen, IGF-1, and H2S on smooth muscle cell proliferation
H<sub>2</sub>S-stimulated bioenergetics in chicken erythrocytes and the underlying mechanism
Correction: Calcium sensing receptor protects high glucose-induced energy metabolism disorder via blocking gp78-ubiquitin proteasome pathway
EXOGENOUS H2S CONTRIBUTES TO RECOVERY OF ISCHEMIC POST-CONDITIONING-INDUCED CARDIOPROTECTION VIA INCREASE OF AUTOPHAGY IN THE AGED HEARTS
Calcium sensing receptor protects high glucose-induced energy metabolism disorder via blocking gp78-ubiquitin proteasome pathway
Exogenous H2S restores ischemic post-conditioning-induced cardioprotection through inhibiting endoplasmic reticulum stress in the aged cardiomyocytes
Involvement of exogenous H2S in recovery of cardioprotection from ischemic post-conditioning via increase of autophagy in the aged hearts
Exogenous H2S contributes to recovery of ischemic post-conditioning-induced cardioprotection by decrease of ROS level via down-regulation of NF-κB and JAK2-STAT3 pathways in the aging cardiomyocytes
The interaction of estrogen and CSE/H<sub>2</sub>S pathway in the development of atherosclerosis
Mediation of exogenous hydrogen sulfide in recovery of ischemic post-conditioning-induced cardioprotection via down-regulating oxidative stress and up-regulating PI3K/Akt/GSK-3β pathway in isolated aging rat hearts
Exogenous hydrogen sulfide restores cardioprotection of ischemic post-conditioning via inhibition of mPTP opening in the aging cardiomyocytes
Interaction of H<sub><b>2</b></sub>S with Calcium Permeable Channels and Transporters
Exogenous spermine inhibits the proliferation of human pulmonary artery smooth muscle cells caused by chemically-induced hypoxia via the suppression of the ERK1/2- and PI3K/AKT-associated pathways
Involvement of calcium-sensing receptors in hypoxia-induced vascular remodeling and pulmonary hypertension by promoting phenotypic modulation of small pulmonary arteries
Mediation of dopamine D2 receptors activation in post-conditioning-attenuated cardiomyocyte apoptosis
Oxygen-sensitive mitochondrial accumulation of cystathionine β-synthase mediated by Lon protease
Involvement of dopamine D2 receptors activation in ischemic post-conditioning-induced cardioprotection through promoting PKC-ε particulate translocation in isolated rat hearts
The expression of calcium-sensing receptor in mouse embryonic stem cells (mESCs) and its influence on differentiation of mESC into cardiomyocytes
Hydrogen sulfide (H <sub>2</sub> S) metabolism in mitochondria and its regulatory role in energy production
Hydrogen sulfide inhibits the translational expression of hypoxia‐inducible factor‐1α
Interaction of Hydrogen Sulfide and Estrogen on the Proliferation of Vascular Smooth Muscle Cells
Exogenous hydrogen sulfide attenuates diabetic myocardial injury through cardiac mitochondrial protection