Area of research
Surgery · Endocrine and Autonomic Systems
Research interest
Research interests include Pancreatic function and diabetes, Circadian rhythm and melatonin, Diabetes and associated disorders, and Dietary Effects on Health.
Melatonin improves postprandial hypoglycemia: an unusual presentation of insulinoma.
Emerin is an effector of oncogenic KRAS-driven nuclear dynamics in pancreatic cancer
ST8Sia6 overexpression protects pancreatic β cells from spontaneous autoimmune diabetes in nonobese diabetic mice
Heterogeneous enhancer states orchestrate β cell responses to metabolic stress.
Diabetes-associated Genetic Variation in <i>MTNR1B</i> and Its Effect on Islet Function.
Glucagon-like peptide-1 receptor blockade impairs islet secretion and glucose metabolism in humans
A transcriptomics-guided drug target discovery strategy identifies receptor ligands for lung regeneration.
The nuclear receptor REV-ERBα is implicated in the alteration of β-cell autophagy and survival under diabetogenic conditions.
Induction of Core Circadian Clock Transcription Factor Bmal1 Enhances β-Cell Function and Protects Against Obesity-Induced Glucose Intolerance.
Time-restricted feeding prevents deleterious metabolic effects of circadian disruption through epigenetic control of β cell function
Time-restricted feeding prevents deleterious metabolic effects of circadian disruption through epigenetic control of β cell function.
Live-cell imaging of glucose-induced metabolic coupling of β and α cell metabolism in health and type 2 diabetes
Insulin Pulse Characteristics and Insulin Action in Non-diabetic Humans.
Accelerated osteocyte senescence and skeletal fragility in mice with type 2 diabetes
Proinflammatory Cytokine Interleukin 1β Disrupts β-cell Circadian Clock Function and Regulation of Insulin Secretion
Diabetes-associated genetic variation in TCF7L2 alters pulsatile insulin secretion in humans
Diabetes-associated genetic variation in TCF7L2 alters pulsatile insulin secretion in humans.
It's about time: clocks in the developing lung.
mTORC1-to-AMPK switching underlies β cell metabolic plasticity during maturation and diabetes
mTORC1 to AMPK switching underlies β-cell metabolic plasticity during maturation and diabetes.
Bmal1 is required for beta cell compensatory expansion, survival and metabolic adaptation to diet-induced obesity in mice
DNA methylation directs functional maturation of pancreatic β cells
Activation of Melatonin Signaling Promotes β-Cell Survival and Function
Reciprocal Regulation of Hepatic and Adipose Lipogenesis by Liver X Receptors in Obesity and Insulin Resistance
Consequences of Exposure to Light at Night on the Pancreatic Islet Circadian Clock and Function in Rats
β-Cell Failure in Type 2 Diabetes: A Case of Asking Too Much of Too Few?
Chronic GLP-1 Receptor Activation by Exendin-4 Induces Expansion of Pancreatic Duct Glands in Rats and Accelerates Formation of Dysplastic Lesions and Chronic Pancreatitis in the KrasG12D Mouse Model
Pulsatile Portal Vein Insulin Delivery Enhances Hepatic Insulin Action and Signaling