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Glenn S. Cowley

Massachusetts Institute of Technology · US
🔎 Find collaborators in Molecular Biology · Pulmonary and Respiratory Medicine →
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Area of research
Molecular Biology · Pulmonary and Respiratory Medicine
Research interest
Research interests include Biology, Cancer research, Gene, Computational biology, Cancer, and CRISPR.
h-index
citations
12,003
works
32
NIH funding
primary concept
email

Recent publications

Genetic barcoding systematically compares genes in del(5q) MDS and reveals a central role for <i>CSNK1A1</i> in clonal expansion
Blood Advances 2022cited by 10position: middledoi
Small-molecule targeting of brachyury transcription factor addiction in chordoma
Nature Medicine 2019cited by 164position: middledoi
Neuronal differentiation and cell-cycle programs mediate response to BET-bromodomain inhibition in MYC-driven medulloblastoma
Nature Communications 2019cited by 60position: middledoi
Gene-centric functional dissection of human genetic variation uncovers regulators of hematopoiesis
eLife 2019cited by 25position: middledoi
Cells Lacking the <i>RB1</i> Tumor Suppressor Gene Are Hyperdependent on Aurora B Kinase for Survival
Cancer Discovery 2018cited by 176position: middledoi
Defining a Cancer Dependency Map
Cell 2017cited by 3,422position: middledoi
Computational correction of copy number effect improves specificity of CRISPR–Cas9 essentiality screens in cancer cells
Nature Genetics 2017cited by 2,136position: middledoi
CRISPR-Cas9 screen reveals a MYCN-amplified neuroblastoma dependency on EZH2
Journal of Clinical Investigation 2017cited by 159position: middledoi
Functional screen of MSI2 interactors identifies an essential role for SYNCRIP in myeloid leukemia stem cells
Nature Genetics 2017cited by 106position: middledoi
Copy-number and gene dependency analysis reveals partial copy loss of wild-type SF3B1 as a novel cancer vulnerability
eLife 2017cited by 84position: middledoi
<i>PIK3CA</i> mutant tumors depend on oxoglutarate dehydrogenase
Proceedings of the National Academy of Sciences 2017cited by 51position: middledoi
HIF activation causes synthetic lethality between the <i>VHL</i> tumor suppressor and the <i>EZH1</i> histone methyltransferase
Science Translational Medicine 2017cited by 46position: middledoi
PRMT1-Mediated Translation Regulation Is a Crucial Vulnerability of Cancer
Cancer Research 2017cited by 41position: middledoi
Abstract 1953: Accelerating prediction of pediatric and rare cancer vulnerabilities using next-generation cancer models
Cancer Research 2017cited by 0position: middledoi
<i>MTAP</i> deletion confers enhanced dependency on the PRMT5 arginine methyltransferase in cancer cells
Science 2016cited by 636position: middledoi
Genomic Copy Number Dictates a Gene-Independent Cell Response to CRISPR/Cas9 Targeting
Cancer Discovery 2016cited by 630position: middledoi
Core Circadian Clock Genes Regulate Leukemia Stem Cells in AML
Cell 2016cited by 276position: middledoi
Integrated genetic and pharmacologic interrogation of rare cancers
Nature Communications 2016cited by 56position: middledoi
Abstract 4367: Accelerating prediction of tumor vulnerabilities using next-generation cancer models
Cancer Research 2016cited by 0position: middledoi
Functional, chemical genomic, and super-enhancer screening identify sensitivity to cyclin D1/CDK4 pathway inhibition in Ewing sarcoma
Oncotarget 2015cited by 98position: middledoi
Complementary genomic approaches highlight the PI3K/mTOR pathway as a common vulnerability in osteosarcoma
Proceedings of the National Academy of Sciences 2014cited by 498position: middledoi
Parallel genome-scale loss of function screens in 216 cancer cell lines for the identification of context-specific genetic dependencies
Scientific Data 2014cited by 413position: firstdoi
In vivo discovery of immunotherapy targets in the tumour microenvironment
Nature 2014cited by 237position: middledoi
The Master Regulator of the Cellular Stress Response (HSF1) Is Critical for Orthopoxvirus Infection
PLoS Pathogens 2014cited by 55position: middledoi
( <i>R</i> )-2-Hydroxyglutarate Is Sufficient to Promote Leukemogenesis and Its Effects Are Reversible
Science 2013cited by 707position: middledoi
A Genome-Scale RNA Interference Screen Implicates NF1 Loss in Resistance to RAF Inhibition
Cancer Discovery 2013cited by 348position: middledoi
SQSTM1 Is a Pathogenic Target of 5q Copy Number Gains in Kidney Cancer
Cancer Cell 2013cited by 165position: middledoi
In Vivo RNAi Screening Identifies a Leukemia-Specific Dependence on Integrin Beta 3 Signaling
Cancer Cell 2013cited by 163position: middledoi
β-Catenin-Driven Cancers Require a YAP1 Transcriptional Complex for Survival and Tumorigenesis
Cell 2012cited by 766position: middledoi
Cancer Vulnerabilities Unveiled by Genomic Loss
Cell 2012cited by 238position: middledoi

Grants

No grants ingested yet.

Frequent collaborators

David E. Root · Broad Institute15 papers (2012–2022)Aviad Tsherniak · Massachusetts Institute of Technology8 papers (2012–2017)Barbara A. Weir · Weatherford College8 papers (2012–2017)William C. Hahn · University of California System7 papers (2012–2017)Francisca Vázquez · Universidade de Santiago de Compostela6 papers (2012–2017)Benjamin L. Ebert · Harvard University5 papers (2012–2022)Rebekka K. Schneider · Harvard University3 papers (2013–2022)Jill P. Mesirov · University of California San Diego3 papers (2012–2012)Marie McConkey · Dana-Farber Cancer Institute3 papers (2016–2022)William G. Kaelin · Harvard University3 papers (2013–2017)Jesse S. Boehm · Massachusetts Institute of Technology3 papers (2012–2017)Rameen Beroukhim · Broad Institute3 papers (2012–2013)Levi A. Garraway · Cancer Research Center3 papers (2013–2017) · 2 papers (2013–2017)Steven E. Schumacher · NorthShore University HealthSystem2 papers (2012–2012)Peter G. Miller · Brigham and Women's Hospital2 papers (2012–2016) · 2 papers (2013–2017)Todd R. Golub · Broad Institute2 papers (2016–2017)Joshiawa Paulk · Novartis (United States)2 papers (2016–2017)Pablo Tamayo · University of San Diego2 papers (2012–2013)
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