Area of research
Pathology and Forensic Medicine · Neurology
Research interest
Research interests include Multiple Sclerosis Research Studies, Neuroinflammation and Neurodegeneration Mechanisms, Peripheral Neuropathies and Disorders, and Neurogenesis and neuroplasticity mechanisms.
A disease-specific convergence of host and Epstein–Barr virus genetics in multiple sclerosis
Blood and CSF biomarkers for multiple sclerosis: emerging clinical applications
Intrathecal Inflammatory Profile and Gray Matter Damage Predict Progression Independent of Relapse Activity in Early Multiple Sclerosis
Mapping Molecular Pathways of Multiple Sclerosis: A Gene Prioritization and Network Analysis of White Matter Pathology Transcriptomics
Determinants and Biomarkers of Progression Independent of Relapses in Multiple Sclerosis
snRNA-seq stratifies multiple sclerosis patients into distinct white matter glial responses
Neuropathological and cerebrospinal fluid correlates of choroid plexus inflammation in progressive multiple sclerosis
Characterization of cortico-meningeal translocator protein expression in multiple sclerosis
Meningeal inflammation as a driver of cortical grey matter pathology and clinical progression in multiple sclerosis
Myeloid cell iron uptake pathways and paramagnetic rim formation in multiple sclerosis
Comparing the Pathology, Clinical, and Demographic Characteristics of Younger and Older‐Onset Multiple Sclerosis
High Levels of Perivascular Inflammation and Active Demyelinating Lesions at Time of Death Associated with Rapidly Progressive Multiple Sclerosis Disease Course: A Retrospective Postmortem Cohort Study
“Ependymal‐in” Gradient of Thalamic Damage in Progressive Multiple Sclerosis
The association between neurodegeneration and local complement activation in the thalamus to progressive multiple sclerosis outcome
B cell rich meningeal inflammation associates with increased spinal cord pathology in multiple sclerosis
The <scp>CSF</scp> Profile Linked to Cortical Damage Predicts Multiple Sclerosis Activity
The Vascular Side of Chronic Bed Rest: When a Therapeutic Approach Becomes Deleterious
Meningeal inflammation changes the balance of TNF signalling in cortical grey matter in multiple sclerosis
Inflammatory intrathecal profiles and cortical damage in multiple sclerosis
Meningeal inflammation and cortical demyelination in acute multiple sclerosis
The cortical damage, early relapses, and onset of the progressive phase in multiple sclerosis
Programmed death 1 is highly expressed on <scp>CD</scp>8<sup>+</sup><scp>CD</scp>57<sup>+</sup> T cells in patients with stable multiple sclerosis and inhibits their cytotoxic response to Epstein–Barr virus
Cognitive impairment predicts disability progression and cortical thinning in MS: An 8-year study
Epilepsy in multiple sclerosis: The role of temporal lobe damage
Regional Distribution and Evolution of Gray Matter Damage in Different Populations of Multiple Sclerosis Patients
Cortical grey matter demyelination can be induced by elevated pro-inflammatory cytokines in the subarachnoid space of MOG-immunized rats
Meningeal inflammation plays a role in the pathology of primary progressive multiple sclerosis