Area of research
Genetics · Endocrinology, Diabetes and Metabolism
Research interest
Research interests include Pathology, Medicine, Biology, Multiple sclerosis, Inflammation, and Pituitary tumors.
Axonal injury is a targetable driver of glioblastoma progression
Preanalytical variables and analytes in liquid biopsy approach for brain tumors: A comprehensive review and recommendations from the RANO Group and the Brain Liquid Biopsy Consortium
The World Health Organization classifications of pituitary neuroendocrine tumours: a clinico-pathological appraisal
High Levels of Perivascular Inflammation and Active Demyelinating Lesions at Time of Death Associated with Rapidly Progressive Multiple Sclerosis Disease Course: A Retrospective Postmortem Cohort Study
Correction to: Amplification of the PLAG-family genes—PLAGL1 and PLAGL2—is a key feature of the novel tumor type CNS embryonal tumor with PLAGL amplification
Data from Levels of p27 Sensitize to Dual PI3K/mTOR Inhibition
Amplification of the PLAG-family genes—PLAGL1 and PLAGL2—is a key feature of the novel tumor type CNS embryonal tumor with PLAGL amplification
Succinate dehydrogenase and MYC-associated factor X mutations in pituitary neuroendocrine tumours
OTHR-41. Amplification of the PLAG family genes – PLAGL1 and PLAGL2 – is a key feature of a novel embryonal CNS tumor type
Concomitant neurodegenerative pathologies contribute to the transition from mild cognitive impairment to dementia
Pituitary neuroendocrine tumors: a model for neuroendocrine tumor classification
The blood–CSF–brain route of neurological disease: The indirect pathway into the brain
How to Classify Pituitary Neuroendocrine Tumors (PitNET)s in 2020
B cell rich meningeal inflammation associates with increased spinal cord pathology in multiple sclerosis
Meningeal inflammation changes the balance of TNF signalling in cortical grey matter in multiple sclerosis
A standardised diagnostic approach to pituitary neuroendocrine tumours (PitNETs): a European Pituitary Pathology Group (EPPG) proposal
Pituitary neuroendocrine tumors (PitNETs): nomenclature evolution, not clinical revolution
From pituitary adenoma to pituitary neuroendocrine tumor (PitNET): an International Pituitary Pathology Club proposal
Kinetic modelling of [ <sup>11</sup> C]PBR28 for 18 kDa translocator protein PET data: A validation study of vascular modelling in the brain using XBD173 and tissue analysis
Complex regulation of neutrophil-derived MMP-9 secretion in central nervous system tuberculosis
Germline or somatic GPR101 duplication leads to X-linked acrogigantism: a clinico-pathological and genetic study
TSPO expression in brain tumours: is TSPO a target for brain tumour imaging?
Somatic<i>GPR101</i>Duplication Causing X-Linked Acrogigantism (XLAG)—Diagnosis and Management
AIP mutations in young patients with acromegaly and the Tampico Giant: the Mexican experience
Cortical Lewy bodies and Aβ burden are associated with prevalence and timing of dementia in Lewy body diseases
The 18-kDa Mitochondrial Translocator Protein in Human Gliomas: An <sup>11</sup>C-(<i>R</i>)PK11195 PET Imaging and Neuropathology Study
Heterogeneous Genetic Background of the Association of Pheochromocytoma/Paraganglioma and Pituitary Adenoma: Results From a Large Patient Cohort
Extensive grey matter pathology in the cerebellum in multiple sclerosis is linked to inflammation in the subarachnoid space
[11C]-(R)PK11195 tracer kinetics in the brain of glioma patients and a comparison of two referencing approaches
Meningeal inflammation plays a role in the pathology of primary progressive multiple sclerosis